Results 171 to 180 of about 712,559 (364)
Advanced Science, EarlyView.TRPA1+αCGRP+ sensory neurons in the nodose ganglion detect external insults such as lipopolysaccharide (LPS) and interact directly with pulmonary neuroendocrine cells (PNECs), promoting their activation and proliferation. This neural‐epithelial interaction amplifies lung inflammation.
Jie Chen +16 more
wiley +1 more source
‘This ongoing innovation continues to push the boundaries of what is possible in dental care' [PDF]
BDJ In Practice
openalex +1 more source
Advanced Science, EarlyView.This study identifies platelet‐derived extracellular vesicles as key immunometabolic regulators in COVID‐19. The delivery of tRF‐His‐GTG‐1 to neutrophils activates TLR8mTOR signaling, disrupts lipophagy, and amplifies NET‐mediated inflammation. Importantly, targeting this axis restores neutrophil homeostasis, offering a potential therapeutic strategy ...
Tsai‐Ling Liao +5 more
wiley +1 more source
Are dental care providers in Saudi Arabia prepared to treat patients with special needs?
Journal of Multidisciplinary Healthcare, 2019 Arwa Alumran, Luloah Almulhim, Bainah Almolhim, Shouq Bakodah, Hessa Aldossary, Saja A AlrayesHealth Information Management and Technology Department, College of Public Health, Imam Abdulrahman bin Faisal University, Alkhobar, Saudi ArabiaBackground ...
Alumran A +5 more
doaj
Dental Oncology: A Pressing Necessity for Comprehensive Cancer Care [PDF]
Ashwini Nerkar Rajbhoj, A. Rajbhoj
openalex +1 more source
DEL‐1 is an Endogenous Senolytic Protein that Inhibits Senescence‐Associated Bone Loss
Advanced Science, EarlyView.Senescent bone marrow stromal cells accumulate in the aging bone microenvironment, promoting bone degeneration. DEL‐1, an endogenous secreted protein, acts as a natural senolytic that selectively eliminates these cells. By engaging a β3 integrin/CD73/adenosine/p38 MAPK/BCL‐2 pathway, DEL‐1 counters aging‐related bone loss, revealing promising ...
Jong‐Hyung Lim +11 more
wiley +1 more source