Results 211 to 220 of about 1,715,754 (387)
A Newly Designed Resonance Frequency Analysis Device for Dental Implant Stability Detection
Wei‐Jen Chang +6 more
openalex +2 more sources
A flexible electrochemical sensor platform can enable continuous, real‐time monitoring of salivary glucose for applications confined to spaces such as the oral cavity. Surface modifications with Prussian blue, chitosan, and zwitterionic polymers enhance sensitivity, selectivity, and antiadherent performance.
Stephanie Klinghammer +8 more
wiley +1 more source
Endoscopic view of antrochoanal polyp with a dental implant [PDF]
Hwang Chul Shin, Jong Seung Kim
openalex +1 more source
CD207+ dendritic cells (DCs) drive emphysema by promoting CD8⁺ T cell cytotoxicity via Birbeck granule‐dependent MHC‐I antigen presentation. This DC subset is expanded by cigarette smoke‐induced oxidative stress, which triggers granulocyte‐macrophage colony‐stimulating factor (GM‐CSF) release from airway epithelium.
Shurui Xuan +10 more
wiley +1 more source
Background Accurate occlusal registration is critical for successful dental implant treatments. Traditional mechanical articulation provides insight into occlusal relationships but cannot replicate complex mandibular movements accurately.
Bundhit Jirajariyavej +5 more
doaj +1 more source
Retrospective analysis of dental implant fracture following loading: A retrospective clinical study [PDF]
Mahmood Abu Ruja +4 more
openalex +1 more source
In this study, we find CD55+ neutrophils show activated NETosis within bone marrow, induce BMSC senescence and osteogenesis inhibition, finally leading to bone aging initiation. Mechanistically, ROS synergizes with the CD55‐driven HIF1α‐PADI4 pathway to promote NETosis.
Yutong Guo +6 more
wiley +1 more source
Mandibular regeneration after immediate load dental implant in a periodontitis patient
Maurizio Catinari +5 more
openalex +1 more source
TRPA1+αCGRP+ sensory neurons in the nodose ganglion detect external insults such as lipopolysaccharide (LPS) and interact directly with pulmonary neuroendocrine cells (PNECs), promoting their activation and proliferation. This neural‐epithelial interaction amplifies lung inflammation.
Jie Chen +16 more
wiley +1 more source

