Results 151 to 160 of about 4,644 (193)

Nephrogenic Diabetes Insipidus

Radiology, 1966
The urographic picture of marked generalized dilatation of the urinary collecting system can present a challenging problem to both the radiologist and the referring clinician. The possible causes are numerous. The frequent occurrence of mild-to-moderate bilateral hydroureter and hydronephrosis during pregnancy or as a consequence of long-standing ...
S S, Miller, M C, Winston
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NEPHROGENIC DIABETES INSIPIDUS

Pediatrics, 1955
1. Two cases of congenital diabetes insipidus resistant to pitressin, or diabetes insipidus of the nephrogenic type, occurring in male cousins during infancy have been described in which the most striking manifestations were recurrent pyrexia, polyuria, polydipsia, poor weight gain and development and hyperelectrolytemia. 2.
J R, WEST, J G, KRAMER
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Nephrogenic diabetes insipidus

Current Opinion in Nephrology and Hypertension, 2000
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Deen, P.M.T., Marr, N., Kamsteeg, E.J., Balkom, B.W.M. van   +3 more
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Nephrogenic Diabetes Insipidus

Annual Review of Physiology, 2001
▪ Abstract  Nephrogenic diabetes insipidus, which can be inherited or acquired, is characterized by an inability to concentrate urine despite normal or elevated plasma concentrations of the antidiuretic hormone arginine vasopressin. Polyuria, with hyposthenuria, and polydipsia are the cardinal clinical manifestations of the disease.
J P, Morello, D G, Bichet
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Nephrogenic Diabetes Insipidus in Children

Acta Paediatrica, 1955
SummaryThe authors describe eight cases of nephrogenic diabetes insipidus in children, six of them studied at a hospital. Five of the cases came from four generations of a family showing a pattern typical of sex‐linked inheritance, two are brothers from another family, and the eighth is a sporadic case.
A, ELLBORG, H, FORSSMAN
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Nephrogenic Diabetes Insipidus With Fluorosis

Pediatrics, 1974
The efficacy of fluoride in the community drinking water for the prevention of dental caries has been well established. The average daily intake of fluids in normal children varies with age, sex, temperature and season. Children with polydipsia as in nephrogenic diabetes insipidus consume excessive quantities of water.
L W, Greenberg, C E, Nelsen, N, Kramer
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A female with X‐linked Nephrogenic diabetes insipidus in a family with inherited central diabetes Insipidus: Case report and review of the literature [PDF]

American Journal of Medical Genetics, Part A, 2020
There are two forms of diabetes insipidus, central (neurohypophyseal), and nephrogenic, caused by pathogenic variants in the AVP gene and the AVPR2 or AQP2 genes, respectively. We report on a four-generation family, seven individuals had central diabetes
Can Ding
exaly   +3 more sources

Intelligence in Nephrogenic Diabetes Insipidus

Archives of Pediatrics & Adolescent Medicine, 1963
Introduction Nephrogenic diabetes insipidus has been cited as one of the causes of mental retardation. Since the course of the disease and the severity of the symptoms can be influenced by the nature of the diet, the intake of water, and more recently by drug therapy, information concerning psychological development in this disease is of importance. A
A L, RUESS, I M, ROSENTHAL
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Nephrogenic Diabetes Insipidus in a Dog

Journal of the American Veterinary Medical Association, 1973
SUMMARY Nephrogenic diabetes insipidus in a 13-week-old male German Shepherd Dog was characterized by polyuria, nocturia, urine of low specific gravity, polydipsia, and loss of body weight. The diagnosis was made on the basis of eliminating pituitary diabetes insipidus, psychogenic polydipsia, and primary renal disease from the differential diagnosis ...
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Nephrogenic Diabetes Insipidus

2009
Publisher Summary This chapter provides details on the role of aquaporin-2 (AQP2), the AVP-dependent water channel of the collecting duct, in several diseases. Arginine vasopressin (AVP) is the hormone that controls serum osmolality by decreasing free water clearance; any condition that interferes with AVP production, secretion and binding to V2 ...
Nine V. A. M. Knoers, Elena N. Levtchenko   +1 more
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