Results 161 to 170 of about 1,945 (206)
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[Dysfibrinogenemia. A new case: dysfibrinogenemia Giessen III (author's transl)].
Klinische Wochenschrift, 1977A new case of dysfibrinogenemia is reported which shows no signs of a haermorrhagic diathesis (dysfibrinogenemia Giessen III). The abnormal fibrinogen was detected by only slight but characteristic alterations of some parameter of the coagulation analysis (prolonged clotting times after addition of thrombin, Reptilase and thrombin coagulase; low ...
F R, Matthias +4 more
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Dysfibrinogenemia and thrombosis.
Nouvelle revue francaise d'hematologie, 1992A thrombotic tendency (venous or arterial) has been reported in some cases of dysfibrinogenemia. We report here the mechanism by which these thrombosis may occur. It may be related either to a defective clot lysis due to a poor reactivity toward fibrinolytic enzymes or to a defective thrombin binding capacity of the abnormal clot.
J, Soria +5 more
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Dysfibrinogenemia and placental abruption
Obstetrics & Gynecology, 2000R Z, Edwards, A, Rijhsinghani
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[Obstetrical management in dysfibrinogenemia].
Geburtshilfe und Frauenheilkunde, 1990Dysfibrinogenaemia is an inherited disorder, characterised by a plasmafibrinogen in normal concentration, but of abnormal quality due to impaired release of fibrinopeptides and/or impaired aggregation of fibrinomonomers. The typical abnormalities of the laboratory findings will be demonstrated by presenting the case reports of 4 patients with 5 ...
H, Heidegger, M, Spannagel, W, Schramm
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Fibrinogen and dysfibrinogenemia.
Annals of clinical and laboratory science, 1981The fibrinogen molecule is becoming increasingly understood. Amino acid sequencing has been undertaken and studies of abnormal fibrinogens are leading to a more functional concept of its structure. Acquired dysfibrinogenemia appears to be a more common problem than previously thought, and may be found in patients with liver disease, cancer ...
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Dysfibrinogenemia as Thrombophilic Factor
1992The prevalence of congenital disfibrinogenemia in venous thrombosis is approximately 0.5% as deduced from 5 studies on familial thrombophilia including a total of 1871 patients [1-5]. This is low in comparison with the prevalence of deficiencies of anticoagulation factors such as antithrombin III protein S and protein C, which together account for one ...
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