Results 261 to 270 of about 436,268 (347)

Atrial Fibroblasts‐Derived Extracellular Vesicles Exacerbate Atrial Arrhythmogenesis

open access: yesAdvanced Science, EarlyView.
Exosome miR‐224‐5p derived from angiotensin II‐treated atrial fibroblasts creates a substrate for AF by promoting atrial electrical remodeling. Increased exosome miR‐224‐5p enhances AF susceptibility by inhibiting CACNA1c expression and decreasing ICa current of atrial cardiomyocytes.
Yue Yuan   +13 more
wiley   +1 more source

Achieving Anti‐Disproportionation Performance Enhancement and Distorted Inverse‐Disproportionation Reaction Correction of Zr2Fe‐Based Hydrogen Isotope Storage Alloys via Element Substitution

open access: yesAdvanced Science, EarlyView.
Undesirable phase transition behavior and anti‐disproportionation performance limit the hydrogen isotope storage of Zr2Fe alloy. With theoretical screening and experiments, partial Cu/Ni substitutions achieve full reversibility and significantly improve anti‐disproportionation properties.
Zhiyi Yang   +12 more
wiley   +1 more source

FOXM1 Protects Against Myocardial Ischemia‐Reperfusion Injury in Rodent and Porcine Models by Suppressing MKRN1‐Dependent LKB1 Ubiquitination

open access: yesAdvanced Science, EarlyView.
FOXM1 maintains mitochondrial bioenergetic function by inhibiting MKRN1‐mediated ubiquitination of LKB1 in cardiomyocytes. Loss of FOXM1 in cardiomyocytes results in upregulation of MKRN1, which enhances LKB1 ubiquitination and disrupts AMPK signaling and energy metabolism pathways. Conversely, FOXM1 overexpression preserves mitochondrial bioenergetics
Shuai Song   +17 more
wiley   +1 more source

Myocardial Proteome in Human Heart Failure With Preserved Ejection Fraction. [PDF]

open access: yesJ Am Heart Assoc
Jani VP   +12 more
europepmc   +1 more source

Home - About - Disclaimer - Privacy