Results 221 to 230 of about 55,295 (265)
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ENDOTOXEMIA IN HUMAN SEPTIC SHOCK
Critical Care Medicine, 1988To evaluate the incidence, pattern and clinical importance of endotoxemia in septic shock, frequent, serial endotoxin determinations were made prospectively in patients with shock. Detectable endotoxin occurred in 43 of 100 patients with septic shock, but in only one of ten patients with shock due to nonseptic causes.
R L, Danner +5 more
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Vascular Lesions in Endotoxemia
1972In the past, vascular lesions in animals injected with endotoxin have been considered to result from anoxia secondary to occlusive thrombotic deposits in vessels. The thrombosis was thought to be caused by activation of clotting factors and platelet procoagulant activity in the flowing blood, with subsequent trapping of fibrin and platelet emboli in ...
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Clinical Forms of Endotoxemia in Burns
Scandinavian Journal of Plastic and Reconstructive Surgery, 1979Two groups of patients in whom endotoxemia was suspected were studied with respect to the laboratory data, clinical picture and autopsy findings. The first group showed the signs of gram-negative septicemia (age range 3 to 54 years with burns of 25 to 80% TBSA) and the second group (age range 20 to 76 years with burns not greater than 20% TBSA) showed ...
R, Königová, Z, Konícková, I, Bouska
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2014
Endotoxemia markedly modifies glutamine metabolism in several tissues with a decrease of intestinal glutamine uptake and metabolism, and decreased oxygen consumption. In liver, endotoxemia decreases glutamine content and mitochondrial oxygen consumption while in skeletal muscles, it increases glutamine synthesis and release resulting in decreased ...
François Blachier +6 more
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Endotoxemia markedly modifies glutamine metabolism in several tissues with a decrease of intestinal glutamine uptake and metabolism, and decreased oxygen consumption. In liver, endotoxemia decreases glutamine content and mitochondrial oxygen consumption while in skeletal muscles, it increases glutamine synthesis and release resulting in decreased ...
François Blachier +6 more
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Endotoxemia and neutrophil activation in vivo
American Journal of Physiology-Heart and Circulatory Physiology, 1988There is a growing body of data to suggest that marginated granulocytes mediate much of the pulmonary damage observed during endotoxemia. The mechanism(s) by which endotoxemia initiates neutrophil margination and cytotoxicity remain either controversial or unknown.
M B, Grisham, J, Everse, H F, Janssen
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Tissue oxygenation and perfusion in endotoxemia
American Journal of Physiology-Heart and Circulatory Physiology, 1996Sepsis is believed to induce disturbances in microcirculatory flow and nutrient exchange, which may result in impaired tissue oxygenation. With the use of an established rat model of endotoxemia, voltametric measurements were made of skeletal muscle (tissue) oxygen tension (PtO2) and its response to inspired oxygen concentration (FIO2).
M, Sair +4 more
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Thrombin and leukocyte recruitment in endotoxemia
American Journal of Physiology-Heart and Circulatory Physiology, 2000Because thrombin has been implicated in sepsis, it has been proposed that antithrombin III (AT III) is beneficial due to its anticoagulatory and antiadhesive effects. Using intravital microscopy, we visualized leukocyte-endothelium interactions in postcapillary venules of the feline mesentery exposed to lipopolysaccharide (LPS).
R C, Woodman +3 more
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Endotoxemia and endotoxemia diagnostics
Plastic and Reconstructive Surgery, 1980Koničková Z +3 more
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1987
The fibrinolytic process includes plasminogen cleavage by different types of plasminogen activators leading to plasmin formation. Plasmin degrades fibrin but also cartilage proteoglycan1 and activates latent collagenase2. Besides its role in hemostasis, fibrinolysis may play a role in tissue remodeling and metastasis of tumor cells3,4. The fibrinolytic
A.-M. Dosne, F. Dubor, L. Chedid
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The fibrinolytic process includes plasminogen cleavage by different types of plasminogen activators leading to plasmin formation. Plasmin degrades fibrin but also cartilage proteoglycan1 and activates latent collagenase2. Besides its role in hemostasis, fibrinolysis may play a role in tissue remodeling and metastasis of tumor cells3,4. The fibrinolytic
A.-M. Dosne, F. Dubor, L. Chedid
openaire +1 more source