Results 61 to 70 of about 11,364,271 (247)
Engineering Civil Society [PDF]
Papers originally presented at a conference and ...
WILLIAMS, DG, YOUNG,, T
core
A‐to‐I editing of miRNAs, particularly miR‐200b‐3p, contributes to HGSOC progression by enhancing cancer cell proliferation, migration and 3D growth. The edited form is linked to poorer patient survival and the identification of novel molecular targets.
Magdalena Niemira +14 more
wiley +1 more source
Digital games in engineering education: systematic review and future trends [PDF]
Chioma Udeozor +3 more
openalex +1 more source
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian +10 more
wiley +1 more source
Engineering at San Jose State University, Summer 2008 [PDF]
https://scholarworks.sjsu.edu/engr_news/1006/thumbnail ...
San Jose State University, Charles W. Davidson College of Engineering
core +2 more sources
Dehydration responsive element binding transcription factors and their applications for the engineering of stress tolerance [PDF]
Pradeep K. Agarwal +3 more
openalex +1 more source
Quantum Electrodynamics of Intense Laser-Matter Interactions: A Tool for Quantum State Engineering [PDF]
Philipp Stammer +7 more
openalex +1 more source
YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy +9 more
wiley +1 more source
Engineering at San Jose State University, Fall 2017 [PDF]
https://scholarworks.sjsu.edu/engr_news/1016/thumbnail ...
San Jose State University, Charles W. Davidson College of Engineering
core +5 more sources
Emerging role of ARHGAP29 in melanoma cell phenotype switching
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster +3 more
wiley +1 more source

