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Unlocking Fertility: How Nitric Oxide Pathways Connect Obesity and Reproductive Health-The Role of Bariatric Surgery. [PDF]
Voros C+15 more
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Treatment of endothelial cell dysfunction in atherosclerosis: a new perspective integrating traditional and modern approaches. [PDF]
Yang L, Li X, Ni L, Lin Y.
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Retraction: The C2238/αANP Variant Is a Negative Modulator of Both Viability and Function of Coronary Artery Smooth Muscle Cells. [PDF]
PLOS One Editors.
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The effect of transient sex hormone fluctuations on endothelial function. [PDF]
Turner CG, DuPont JJ.
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Roles of eNOS in atherosclerosis treatment
Inflammation Research, 2019Atherosclerosis (AS) is the main pathogeny of coronary heart disease, cerebral infarction and peripheral vascular disease. Endothelial dysfunction is one of the important pathogenesis of AS. As an important endothelium-derived relaxation factor, nitric oxide (NO) plays a role in cardiovascular protection and anti-AS function; but in the pathological ...
Fen-Fang Hong+6 more
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Pharmacological Prevention of eNOS Uncoupling
Current Pharmaceutical Design, 2014Under physiological conditions, nitric oxide (NO) is produced in the vasculature mainly by the endothelial NO synthase (eNOS). This endothelium-derived NO is a protective molecule with antihypertensive, antithrombotic and anti-atherosclerotic properties.
Huige Li, Ulrich Förstermann
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Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids, 2008
In the mammalian cardiovascular system, nitric oxide (NO), a small diffusible gaseous signal mediator, plays pivotal roles in the maintenance of vascular homeostasis. The endothelial isoform of nitric oxide synthase (eNOS) is activated by diverse agonist-modulated cell surface receptors, and eNOS-derived NO is a key determinant of blood pressure ...
Thomas Michel, Junsuke Igarashi
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In the mammalian cardiovascular system, nitric oxide (NO), a small diffusible gaseous signal mediator, plays pivotal roles in the maintenance of vascular homeostasis. The endothelial isoform of nitric oxide synthase (eNOS) is activated by diverse agonist-modulated cell surface receptors, and eNOS-derived NO is a key determinant of blood pressure ...
Thomas Michel, Junsuke Igarashi
openaire +3 more sources