Results 121 to 130 of about 1,156 (179)
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Enterotoxemia in Two Foals

Journal of the American Veterinary Medical Association, 1978
SUMMARY Two Quarter Horse foals from different premises died from enterotoxemia. Clostridium perfringens toxins α and β were demonstrated in the foals' intestines by mouse protection tests. Clostridium perfringens type C was isolated from the intestines of each foal.
C W, Dickie   +2 more
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Enterotoxemia in Neonatal Calves

Veterinary Clinics of North America: Food Animal Practice, 1985
The incidence, bacterial characteristics, disease syndromes, diagnosis, treatment, and prevention of enterotoxemia of neonatal calves caused by Clostridium perfringens (Types A, B, C, D, and E) are reviewed.
openaire   +2 more sources

Enterotoxemia in Cattle

American Association of Bovine Practitioners Conference Proceedings, 1971
Enterotoxemia is an acute infectious but non-contagious disease caused by the toxins of Clostridium perfringens Type D or C, or both and characterized by sudden death. This disease is often suspected and diagnosed clinically quite frequently in feedlot cattle. A definitive diagnosis is difficult in most cases.
openaire   +1 more source

Enterotoxemia of Small Ruminants

American Association of Bovine Practitioners Conference Proceedings, 2009
Enterotoxemia is a common, frequently fatal disease of small ruminants caused by enteric superinfection with Clostridium perfringens. C. perfringens type C causes hemorrhagic enteritis of lambs. Type D causes colitis in goats and rapidly progressive, multi-organ failure in sheep, characterized by recumbency, convulsions, and opisthotonus. Prevention of
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Enterotoxemia in rabbits.

Laboratory animal science, 1979
The presence of Clostridium perfringens Type E iota toxin was confirmed from the cecal contents of 23 of 46 rabbits which died of enteritis complex. The most consistent lesions observed were hemorrhage and edema in the cecum. Rabbit toxicity tests showed the toxic cecal contents were lethal for young rabbits unless incubated with Clostridium ...
N M, Patton   +3 more
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Gastric dilatation and enterotoxemia in ten captive felids

Journal of the American Veterinary Medical Association, 2018
Abstract CASE DESCRIPTION 10 large felids at 8 facilities were determined or suspected to have developed gastric dilatation with or without enterotoxemia over a 20-year period. Four felids were found dead with no premonitory signs. CLINICAL FINDINGS 4 felids (2 male snow leopards [Uncia uncia], 1 male Amur tiger [Panthera tigris altaica], and 1 male ...
Kadie M, Anderson   +7 more
openaire   +2 more sources

Type A Enterotoxemia in Captive Wild Goats

Journal of the American Veterinary Medical Association, 1970
SUMMARY This report relates the death loss of 20% of a herd of captive wild goats (Capra ibex sibericus) associated with Clostridium perfringens, type A enterotoxemia. All illnesses were diagnosed at necropsy. A rich diet and adverse weather conditions were thought to be predisposing factors.
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Enterotoxemia in small ruminants

American Association of Bovine Practitioners Conference Proceedings, 2019
Enterotoxemia due to toxin production from Clostridium perfringens type C and D is reported to be the most common cause of unnatural death in small ruminants. This session addresses clinical signs, diagnostic challenges, treatment, and vaccination strategies to prevent further cases.
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Fluid Compartment Alterations in the Monkey with Staphylococcic B Enterotoxemia

American Journal of Veterinary Research, 1970
SUMMARY Body fluid compartment measurements were obtained serially in unanesthetized, sitting rhesus monkeys given highly purified staphylococcic enterotoxin B (seb) intravenously (iv) at the dose level of 1 mg./kg. of body weight. All values were compared with those obtained in identical control studies, using the same monkeys, and with those of sham ...
D A, Rhoda, D D, Elsberry, W R, Beisel
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Clostridium perfringens type C enterotoxemia in a newborn foal

Journal of the American Veterinary Medical Association, 1986
A 1-day old, full-term foal with a history of colic died 2 hours after admission. Necropsy revealed an extremely flaccid, fluid-filled intestinal tract. Histopathologically, the superficial intestinal mucosa was completely necrotic, with minimal inflammatory response. Numerous large, gram-positive rods covered the villi.
M, Howard-Martin   +3 more
openaire   +2 more sources

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