Results 81 to 90 of about 3,693 (191)

10. Complex regional pain syndrome

open access: yesPain Practice, Volume 25, Issue 1, January 2025.
Abstract Introduction Complex regional pain syndrome (CRPS) is a clinical disorder that can develop following surgery or trauma. Based on the most prominent underlying pathophysiological mechanisms, CRPS can be classified into different subtypes, namely inflammatory, nociplastic/neuropathic, vasomotor, and motor.
Daniël P. C. van der Spek   +4 more
wiley   +1 more source

Analgesic Effects of GpTx-1, PF-04856264 and CNV1014802 in a Mouse Model of NaV1.7-Mediated Pain [PDF]

open access: yes, 2016
Loss-of-function mutations of NaV1.7 lead to congenital insensitivity to pain, a rare condition resulting in individuals who are otherwise normal except for the inability to sense pain, making pharmacological inhibition of NaV1.7 a promising therapeutic ...
Alewood, P.   +12 more
core   +2 more sources

Acrocyanosis: The Least Known Acrosyndrome Revisited With a Dermatologic Perspective

open access: yesDermatology Research and Practice, Volume 2025, Issue 1, 2025.
Background: Acrocyanosis is a functional peripheral vascular disorder, currently categorized under the canopy of acrosyndromes, i.e., a group of clinically similar and significantly overlapping vascular disorders involving the acral skin. The disorder might be primary or secondary, depending on the cause.
Deniz Demircioğlu   +2 more
wiley   +1 more source

La terapia intratecale nelle vasculopatie - Intrathecal therapy in vascular diseases

open access: yesPathos, 2012
Erythromelalgia is a rare clinical syndrome, characterized by erythema, burning pain and increased skin temperature in the extremities. The therapeutic treatment of this pathology is not yet standardized and both the administration of several drugs or ...
Rosario Russo   +2 more
doaj  

Early- and Late-Onset Inherited Erythromelalgia

open access: yesPediatric Neurology Briefs, 2009
A genotype-phenotype relationship at the clinical, cellular and molecular levels is shown in a case of erythromelalgia of relatively late onset, in a study at Yale University School of Medicine, and centers in China.
J Gordon Millichap
doaj   +1 more source

Diagnosis and initial management of children presenting with premature loss of primary teeth associated with a systemic condition: A scoping review and development of clinical aid

open access: yesInternational Journal of Paediatric Dentistry, Volume 34, Issue 6, Page 871-890, November 2024.
Abstract Background Premature loss of primary teeth (PLPT) can be a rare presentation of systemic medical conditions. Premature loss of primary teeth may present a diagnostic dilemma to paediatric dentists. Aims To identify systemic conditions associated with PLPT and develop a clinical aid.
Claudia Heggie   +3 more
wiley   +1 more source

Analyzing China’s contributions to major dermatologic journals from the past 20 years [PDF]

open access: yes, 2020
Background: Over the past 20 years, China has experienced an increased popularity of Western medicine. The impact of Western medicine in China on the field of dermatology is not well characterized.
Bray, Jeremy K, Feldman, Steven R
core  

Biomolecular and histological features in pediatric essential thrombocythemia: adequacy of who diagnostic criteria [PDF]

open access: yes, 2017
Myeloproliferative neoplasms (MPN), Essential Thrombocythemia (ET), Polycythemia Vera (PV) and primary myelofibrosis (PMF), are clonal disorders of the hematopoietic stem cell.
Bertozzi, Irene
core   +1 more source

Methylation of TET2, CBL and CEBPA in Ph-negative myeloproliferative neoplasms [PDF]

open access: yes, 2010
A loss-of-function mutation of TET2, CBL and CEBPA has been implicated in the pathogenesis or leukaemic transformation of myeloproliferative neoplasm.
Chim, CS, Fung, TK, Wan, TS, Wong, KF
core   +1 more source

Platelets as mediators of Thromboinflammation in chronic Myeloproliferative Neoplasms [PDF]

open access: yes, 2019
Chronic myeloproliferative neoplasms (MPN) are stem cell disorders driven by mutations in JAK2, CALR, or MPL genes and characterized by myeloid proliferation and increased blood cell counts.
Heller, Paula Graciela   +1 more
core   +1 more source

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