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Ethnic Studies Newsletter Vol. 3 No. 1
, 1982 Seven page newsletter from the Ethnic Studies department dated October 1982. Newsletter is Volume 3, Number 1. Newsletter includes Feature on Dr. Herbert Aptheker; Feature on Dr.
Ethnic Studies Department;
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Molecular Oncology, EarlyView.This study shows that lung adenocarcinomas exploit developmental branching morphogenesis to acquire a therapy resistant basal‐like tumour cell state. This process was found to be regulated by combined TP53 loss‐of‐function and type‐I interferon signalling, identifying a novel axis for biomarker and therapeutic target discovery.
Kamila J Bienkowska +13 more
wiley +1 more source
Ethnic Studies Newsletter Vol. 4 No. 1
, 1983 Six page newsletter from the Ethnic Studies department dated October 1983. Newsletter is Volume 4, Number 1. Newsletter includes Features on Dr. Kate Conway McDavis; News Items on Northern California Regional Conference, Research Project on Japanese ...
Ethnic Studies Department;
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Molecular Oncology, EarlyView.Combining osimertinib with the STING agonist ADU‐S100 activates innate and adaptive immunity to overcome the non‐inflamed microenvironment of Egfr‐mutant lung cancer. This combination increases NK and CD8+ T‐cell infiltration, associated with activation of the STING‐IRF3 pathway and local immunogenic cell death.
Jun Nishimura +19 more
wiley +1 more source
On the Theory of Ethnic Conflict [PDF]
We present a theory of ethnic conflict in which coalitions formed along ethnic lines compete for the economy’s resources. The role of ethnicity is to enforce coalition membership: in ethnically homogeneous societies members of the losing coalition can ...
Francesco Caselli +1 more
core +4 more sources
Loss of IGF‐1R impairs DNA‐PKcs recruitment to chromatin leading to defective end‐joining
Molecular Oncology, EarlyView.IGF‐1R promotes radioresistance by facilitating DNA‐PKcs recruitment to chromatin, enabling non‐homologous end‐joining (NHEJ) repair of double‐strand breaks. Inhibition or loss of IGF‐1R disrupts this recruitment to damage sites, driving compensatory reliance on microhomology‐mediated end‐joining (MMEJ) repair.
Matthew O. Ellis +3 more
wiley +1 more source
Bucharest Working Papers in Linguistics, 2007 This paper is a part of a larger project on the language behaviour of different ethnic groups in Shaw’s plays. It focuses on the British ethnic stereotype as it emerges from the playwright’s ideology of the end of the 19th century, and tries to answer ...
Zsuzsanna Ajtony
doaj
Molecular Oncology, EarlyView.We identify USP29 as the only DUB mirroring CA9 expression, a marker of hypoxia and HIF pathway activation associated with PCA aggressiveness. USP29 stabilizes HIF‐1α and HIF‐2α via a noncanonical mechanism that is independent of PHD/pVHL activity yet relies on proteasomal regulation, establishing USP29 as a previously unrecognized regulator of hypoxic
Amelie S Schober +16 more
wiley +1 more source
MITF maintains genome stability in nonmelanocyte lineages
Molecular Oncology, EarlyView.MITF is essential for melanocyte survival and acts as an oncogene in 10%–20% of melanomas. We show that MITF depletion causes genome instability in nonmelanocytic cells, leading to LATS2‐mediated P53 activation, cell cycle arrest, and apoptosis. This study highlights the role of MITF as a genome maintenance factor beyond the melanocyte lineage. Created
Drifa H. Gudmundsdottir +13 more
wiley +1 more source
Ethnic Studies Newsletter Vol. 4 No. 2
, 1983 Seven page newsletter from the Ethnic Studies department dated December 1983. Newsletter is Volume 4, Number 2. Newsletter includes pieces on Dr. Benjamin P.
Ethnic Studies Department;
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