An event-related brain potential correlate of visual short-term memory
Peter Klaver+4 more
openalex +2 more sources
On the locus of the semantic satiation effect: Evidence from event-related brain potentials [PDF]
John Kounios+2 more
openalex +1 more source
Implicit and explicit learning of event sequences: an analysis with event-related brain potentials. [PDF]
Prof. Rösler, Rüsseler, Jascha
core +1 more source
β‐TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1
Low levels of β‐TrCP (Panel A) allow the accumulation of BRCA1 and CtIP, which facilitate the repair of cisplatin‐induced DNA damage via homologous recombination (HR) and promote tumor cell survival. In contrast, high β‐TrCP expression (Panel B) leads to BRCA1 and CtIP degradation, impairing HR repair, resulting in persistent DNA damage and apoptosis ...
Rocío Jiménez‐Guerrero+8 more
wiley +1 more source
Emotion recognition in deaf individuals: evidence from event-related potentials. [PDF]
Kang SM, Apar LS, Hurtado R.
europepmc +1 more source
Auditory attention processing in 5‐year‐old children born preterm: evidence from event‐related potentials [PDF]
Remy Dupin+3 more
openalex +1 more source
Proteomic and phosphoproteomic analyses were performed on lung adenocarcinoma (LUAD) tumors with EGFR, KRAS, or EML4–ALK alterations and wild‐type cases. Distinct protein expression and phosphorylation patterns were identified, especially in EGFR‐mutated tumors. Key altered pathways included vesicle transport and RNA splicing.
Fanni Bugyi+12 more
wiley +1 more source
Sleep efficiency and event-related potentials in patients with depression: the mediating role of serum C-reactive protein. [PDF]
Fang L+8 more
europepmc +1 more source
Resampling approach to statistical inference: Bootstrapping from event-related potentials data [PDF]
Francesco Di Nocera, Fabio Ferlazzo
openalex +1 more source
Targeting of PTP4A3 overexpression sensitises HGSOC cells towards chemotherapeutic drugs
In HGSOC with normal KRAS expression, high PTP4A3 expression regulates autophagy activation. Conversely, in HGSOC with high KRAS expression, KRAS dictates autophagy control, and PTP4A3 is not required. When high PTP4A3 expression is inhibited, HGSOC cells are preferentially sensitised towards DNA‐damaging agents.
Ana López‐Garza+3 more
wiley +1 more source