Results 51 to 60 of about 68,770 (301)

The Impact of Resistance Exercise and Age on Excess Post- Exercise Oxygen Consumption

open access: bronzeMedicine & Science in Sports & Exercise, 2008
Mark G. Cullum, J. W. Yates
openaire   +3 more sources

The power of microRNA regulation—insights into immunity and metabolism

open access: yesFEBS Letters, EarlyView.
MicroRNAs are emerging as crucial regulators at the intersection of metabolism and immunity. This review examines how miRNAs coordinate glucose and lipid metabolism while simultaneously modulating T‐cell development and immune responses. Moreover, it highlights how cutting‐edge artificial intelligence applications can identify miRNA biomarkers ...
Stefania Oliveto   +2 more
wiley   +1 more source

Metabolic responses to strength training: an emphasis on energy expenditure.

open access: yesRevista Brasileira de Cineantropometria e Desempenho Humano, 2011
The present study reviews the results reported in the literature regarding energy expenditure in strength training (ST). For this purpose, a search was conducted in the Medline and Sport Discus databases using the following key words: strength training ...
Ronei Silveira Pinto   +2 more
doaj   +1 more source

GEDAE-LaB: A Free Software to Calculate the Energy System Contributions during Exercise. [PDF]

open access: yesPLoS ONE, 2016
PURPOSE:The aim of the current study is to describe the functionality of free software developed for energy system contributions and energy expenditure calculation during exercise, namely GEDAE-LaB.
Rômulo Bertuzzi   +7 more
doaj   +1 more source

Integrative systems‐level analysis reveals a contextual crosstalk between hypoxia and global metabolism in human breast tumors

open access: yesMolecular Oncology, EarlyView.
Breast tumor samples scored for metabolic deregulation (M1 to M3) were given a hypoxia score (HS). The highest HS occurred in patients with strongest metabolic deregulation (M3), supporting tumor aggressiveness. HS correlated with the highest number of metabolic pathways in M1. This suggests hypoxia to be an early event in metabolic deregulation.
Raefa Abou Khouzam   +2 more
wiley   +1 more source

Chemical Power for Swarms of Microscopic Robots in Blood Vessels [PDF]

open access: yesarXiv, 2023
Microscopic robots in the bloodstream could obtain power from fuel cells using glucose and oxygen. Previous studies of small numbers of such robots operating near each other showed how robots compete with their neighbors for oxygen. However, proposed applications involve billions of such robots operating throughout the body.
arxiv  

Inhibition of acyl‐CoA synthetase long‐chain isozymes decreases multiple myeloma cell proliferation and causes mitochondrial dysfunction

open access: yesMolecular Oncology, EarlyView.
Triacsin C inhibition of the acyl‐CoA synthetase long chain (ACSL) family decreases multiple myeloma cell survival, proliferation, mitochondrial respiration, and membrane potential. Made with Biorender.com. Multiple myeloma (MM) is an incurable cancer of plasma cells with a 5‐year survival rate of 59%.
Connor S. Murphy   +12 more
wiley   +1 more source

Training effects of set- and repetition-interval rest time on recumbent-boxing exercise: Could virtual reality improve further?

open access: yesiScience, 2023
Summary: This study examined the influence of set-interval and repetition-interval rest time of virtual reality (VR) boxing game in supine-lying posture.
Yi Wang   +7 more
doaj  

Polarity-induced oxygen vacancies at LaAlO3|SrTiO3 interfaces [PDF]

open access: yesPhysRevB.82.165127 (2010), 2010
Using first-principles density functional theory calculations, we find a strong position and thickness dependence of the formation energy of oxygen vacancies in LaAlO3|SrTiO3 (LAO|STO) multilayers and interpret this with an analytical capacitor model.
arxiv   +1 more source

Respiratory complex I‐mediated NAD+ regeneration regulates cancer cell proliferation through the transcriptional and translational control of p21Cip1 expression by SIRT3 and SIRT7

open access: yesMolecular Oncology, EarlyView.
NAD+ regeneration by mitochondrial complex I NADH dehydrogenase is important for cancer cell proliferation. Specifically, NAD+ is necessary for the activities of NAD+‐dependent deacetylases SIRT3 and SIRT7, which suppress the expression of p21Cip1 cyclin‐dependent kinase inhibitor, an antiproliferative molecule, at the translational and transcriptional
Masato Higurashi   +5 more
wiley   +1 more source

Home - About - Disclaimer - Privacy