Results 81 to 90 of about 2,329 (305)

Development of TCSC Application to Fault Current Limiters

open access: yesIEEJ Transactions on Power and Energy, 2001
AbstractA new Fault Current Limiter (FCL) is proposed which consists of a conventional Current Limiting Reactor (CLR) and a Thyristor‐Controlled Series Compensator (TCSC) in series. In normal operation, it works to compensate for impedance of the CLR and a line, while it limits short‐circuit currents during system faults.
Yuji Yamazaki   +4 more
openaire   +2 more sources

Operational control and protection implications of fault current limitation in distribution networks

open access: yes, 2009
Rising short-circuit fault current levels is one of the problems associated with the increased presence of distributed generation (DG) in electrical networks.
Blair, S.M.   +3 more
core  

Impact of marine power system architectures on IFEP vessel availability and survivability

open access: yes, 2007
In recent years integrated full electric propulsion (IFEP) has become a popular power system concept within the marine community, both for the naval and the commercial community.
J.D. Schuddebeurs   +7 more
core   +1 more source

CD47 promotes mitogen‐activated protein kinase and epithelial‐to‐mesenchymal transition molecular programs to drive prometastatic phenotypes in non‐small cell lung cancer

open access: yesMolecular Oncology, EarlyView.
Beyond its role in immune evasion, this study identified that CD47 drives tumor‐intrinsic signaling in non‐small cell lung cancer (NSCLC). Transcriptomic profiling and functional studies revealed that CD47 regulates cell adhesion, migration, and metastasis through an ERK–EMT signaling axis.
Asa P.Y. Lau   +8 more
wiley   +1 more source

Heterozygous loss‐of‐function alleles associate the conserved 3′‐5′ exoribonuclease EXOSC10 with hypersensitivity to the anticancer drug 5‐fluorouracil

open access: yesMolecular Oncology, EarlyView.
EXOSC10, an essential nuclear RNA exosome‐associated 3′‐5′ exoribonuclease, is inhibited by the anticancer drug 5‐fluorouracil (5‐FU), and EXOSC10 depletion increases 5‐FU sensitivity. The colon‐cancer variant EXOSC10S402T, located in a proteolysis motif, is stable and nuclear but nonfunctional in vivo.
Radhika Sain   +10 more
wiley   +1 more source

Thyristor based bridge‐type fault current limiter for fault current limiting capability enhancement

open access: yesIET Generation, Transmission & Distribution, 2016
This study deals with a developed thyristor based bridge‐type fault current limiter (TBFCL), in which the main fault current limiting function is relegated to a limiting circuit controlled by a silicon controlled rectifier (SCR). Since SCR is a reliable power electronic switch and can be found in high ratings, the proposed TBFCL can be utilised in ...
Teymoor Ghanbari   +2 more
openaire   +1 more source

Longitudinal circulating tumor DNA profiling in patients with advanced endometrial cancer using an off‐the‐shelf targeted NGS panel

open access: yesMolecular Oncology, EarlyView.
Intratumour heterogeneity complicates precision management of advanced endometrial cancer. Circulating tumor DNA (ctDNA) offers a minimally invasive strategy to capture tumor evolution and therapeutic resistance. Here, we compare tumor‐agnostic NGS with tumor‐informed ddPCR, outlining their relative sensitivity, concordance, and clinical implications ...
Carlos Casas‐Arozamena   +15 more
wiley   +1 more source

Design of a 10kV,500A Inductive Superconducting Fault Current Limiter

open access: yes, 2017
The design aspect of a 10kV,500A inductive superconducting fault current limiter was analyzed. The effect of the superconductor in the fault current limiter was defined. When placed in the system, in the absence of fault current,the superconducting fault
Akejelu, Daniel Limi
core  

Interaction of HS1BP3 with cortactin modulates TKS5 localisation, cell secretion and cancer malignancy

open access: yesMolecular Oncology, EarlyView.
Here, we demonstrate that HS1BP3 interacts with Cortactin through a proline‐rich region (PRR3.1) and show that this interaction, and HS1BP3 itself, promote cancer cell proliferation and invasion. Inhibition of this interaction leads to build‐up of TKS5 in multivesicular endosomes and altered secretion of CD63 and CD9, providing an explanation for the ...
Arja Arnesen Løchen   +9 more
wiley   +1 more source

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