Results 201 to 210 of about 6,491,911 (254)

Atopic dermatitis, primary atopic disorders, and the cutaneous microbiome: current understanding of an expanding field. [PDF]

Front Pediatr
Abidov A, Bayer DK.
europepmc   +1 more source

Conditioned medium from stem cells of human exfoliated deciduous teeth ameliorates mouse ovalbumin-specific allergic dermatitis. [PDF]

Stem Cell Res Ther
Xu Y, Hashimoto N, Kano F, Xia L, Ding C, Lao H, Wang Y, Arai T, Iwasaki T, Yamamoto A.   +9 more
europepmc   +1 more source

Negative feedback regulation of CHK2 in VIN18 raft cultures naturally infected with HPV18. [PDF]

Virus Res
Wu M, Liu J, Li H, Yang R.
europepmc   +1 more source

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Defects of filaggrin-like proteins in both lesional and nonlesional atopic skin

Journal of Allergy and Clinical Immunology, 2013
Atopic dermatitis (AD) is a chronic inflammatory skin disease characterized by a disturbed epidermal barrier. In a subset of patients, this is explained by nonsense mutations in the gene encoding filaggrin (FLG).We sought to evaluate the respective role of FLG mutations and proinflammatory cytokines and to assess the expression of FLG, hornerin (HRNR),
Laurence Pellerin, Marie-Claire Mechin, Elke Rodriguez   +2 more
exaly   +4 more sources

Gene regulation of filaggrin and other skin barrier proteins via aryl hydrocarbon receptor

Journal of Dermatological Science, 2015
Aryl hydrocarbon receptor (AHR) is a ligand-dependent transcription factor that binds to structurally diverse chemicals including dioxins, coal tar, flavonoids and tryptophan photoproducts. Upon ligation, cytoplasmic AHR translocates to the nucleus, heterodimerizes with aryl hydrocarbon receptor nuclear translocator and mediates numerous biological ...
Masutaka Furue, Gaku Tsuji, Chikage Mitoma   +2 more
exaly   +4 more sources

Inflammatory-Driven Depletion of Filaggrin Proteins

, 2014
Filaggrin has become the major research focus within the field of skin barrier abnormality over the last 5–10 years. Null mutations in the (pro)-filaggrin gene (FLG) have been shown to be a major risk factor for the development of atopic dermatitis, a disease in which one of the hallmarks is a decreased skin barrier function.
C. Vestergaard, M. Deleuran
semanticscholar   +3 more sources

Detection of New Epitopes of Antibodies to Filaggrin in Filaggrin Protein Molecule

Bulletin of Experimental Biology and Medicine, 2011
Immunogenic characteristics of filaggrin protein molecule as an antigen for antibodies to filaggrin, markers of early rheumatoid arthritis, were studied. Two new peptide motives, possible epitopes for antibodies to filaggrin, were shown in the filaggrin molecule by predictive analysis using programmed algorithms.
A G, Kobylyansky, A N, Nekrasov, V I, Kozlova, M Yu, Sandin, B A, Alikhanov, V V, Demkin   +5 more
openaire   +2 more sources

Increased expression of Filaggrin and Claudin‐1 in the ocular surface of patients with atopic dermatitis

Journal of the European Academy of Dermatology and Venereology, 2021
Atopic dermatitis (AD) is an itchy, chronic and inflammatory skin condition, with dysfunctional immune response and skin barrier defects. Reduction of filaggrin (FLG) and tight junctions (TJ) proteins, such as claudin‐1 (CLDN‐1), expression in cutaneous ...
Thais Maria Pinheiro Callou, R. L. Orfali, M. Sotto, N. V. Pereira, M. Zaniboni, V. Aoki, Mariane Pereira Brito, M. Matsuda, R. Santo   +8 more
semanticscholar   +1 more source

Expression of filaggrin-2 protein in the epidermis of human skin diseases: A comparative analysis with filaggrin

Biochemical and Biophysical Research Communications, 2014
Filaggrin-2 is a member of the S100 fused-type protein family, and the structural features and expression of filaggrin-2 are similar to those of profilaggrin, a protein essential for keratinization. In the present study, we investigated the expression profile of filaggrin-2 in patients with skin diseases using antibodies against the repetitive region ...
Teruhiko, Makino, Megumi, Mizawa, Takako, Yamakoshi, Mikiro, Takaishi, Tadamichi, Shimizu   +4 more
openaire   +2 more sources