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Effect of cyclosporine on fractional excretion of sodium in renal transplant patients
Clinical Transplantation, 1990Cyclosporine (CSA) nephrotoxicity remains a major concern for clinicians. Experimental animal and human studies indicate that CSA causes an increase in renal vascular resistance, a decrease in renal blood flow and a reduction in glomerular filtration rate (GFR).
Bharat V. Shah +2 more
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Fraction excretion of sodium as a marker of tubular injury – is it sensitive enough?
Clinical chemistry and laboratory medicine, 2011Background. The only promptly avaible testfor early diagnosis of acute kidney failure, so far, is fraction excretion of sodium (FENa). Racent studies indetified neutrophil gelatinase-associated lipocalin (NGAL) as a promising biomarker for such state.
Mandić, Dario +3 more
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Journal of the American Society of Nephrology, 2023
Mohammad O. Abdelhafez +2 more
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Mohammad O. Abdelhafez +2 more
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Low fractional excretion of sodium with contrast media-induced acute renal failure.
Archives of Internal Medicine, 1980L. Fang +3 more
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Clinica chimica acta; international journal of clinical chemistry, 1983
P. Whiting +5 more
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P. Whiting +5 more
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American journal of clinical and experimental urology
Background: Cystic Fibrosis (CF) is a chronic disease associated with low sodium status. The patients are usually treated with oral sodium chloride to control the side effects of low sodium status. Therefore, the fractional excretion of sodium (FENa) was assessed in patients with cystic fibrosis (CF) treated with oral sodium chloride (NaCl).
Majid, Keivanfar +3 more
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Background: Cystic Fibrosis (CF) is a chronic disease associated with low sodium status. The patients are usually treated with oral sodium chloride to control the side effects of low sodium status. Therefore, the fractional excretion of sodium (FENa) was assessed in patients with cystic fibrosis (CF) treated with oral sodium chloride (NaCl).
Majid, Keivanfar +3 more
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Low fractional excretion of sodium in acute renal failure: role of timing of the test and ischemia.
American Journal of Nephrology, 1986F. Brosius, K. Lau
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