Results 21 to 30 of about 122,715 (266)

Heterotopic gastric mucosa leading to chronic jejunal obstruction

open access: yesJournal of Pediatric Surgery Case Reports, 2019
Heterotopic gastric mucosa (HGM) has histologic features of gastric mucosa located outside of the stomach. The congenital form is a rare clinical entity that can present with GI bleeding, intussusception from a polypoid mass or intermittent obstruction ...
Bethany J. Slater   +4 more
doaj   +1 more source

Heterotopic gastric mucosa in the rectum

open access: yesJournal of Pediatric Surgery Case Reports, 2020
Heterotopic gastric mucosa is rare in the pediatric population. We present a case of a 14-year-old girl with a 6-month history of intermittent rectal bleeding and failed sclerotherapy. Excision ultimately demonstrated heterotopic gastric mucosa.
Sairvan Fernandes   +4 more
doaj   +1 more source

Glycophenotypic alterations induced by Pteridium aquilinum in mice gastric mucosa: synergistic effect with Helicobacter pylori infection. [PDF]

open access: yesPLoS ONE, 2012
The bracken fern Pteridium aquilinum is a plant known to be carcinogenic to animals. Epidemiological studies have shown an association between bracken fern exposure and gastric cancer development in humans.
Joana Gomes   +10 more
doaj   +1 more source

Absolute quantification of a plasma tRNA-derived fragment for the diagnosis and prognosis of gastric cancer

open access: yesFrontiers in Oncology, 2023
BackgroundThe transition from a healthy gastric mucosa to gastric cancer is a multi-step process. Early screening can significantly improve the survival rate of gastric cancer patients.
Shuangshuang Zhang   +9 more
doaj   +1 more source

Mechanism of Epithelial-mesenchymal Transition Leading to Gastric Cancer Induced by Helicobacter pylori

open access: yesXiehe Yixue Zazhi
Helicobacter pylori (Hp) is a unipolar, microaerobic, multiflagellar, spiral-shaped Gram-negative bacterium that survives and colonizes human gastric mucosa. As a classⅠcarcinogen associated with gastric cancer, long-term stimulation of gastric mucosa by
SUN Zhencan   +6 more
doaj   +1 more source

Gastric Mucosa Plasma Cells is Unspecific for Diagnosing Helicobacter pylori Infection

open access: yesThe Indonesian Journal of Gastroenterology, Hepatology and Digestive Endoscopy, 2020
Background: A high density of Helicobacter pylori is believed to trigger an accumulation of plasma cells in the gastric mucosa. This accumulation stimulated the production of antibodies, causing high antibodies titer being circulated in the blood.
Gilda Hartecia   +3 more
doaj   +1 more source

Metastatic Calcinosis of Gastric Mucosa [PDF]

open access: yesJournal of Investigative Medicine High Impact Case Reports, 2020
Calcinosis cutis refers to the deposition of calcium salts in the cutaneous and subcutaneous tissue and is frequently associated with inflammation. Gastric calcinosis can be classified into metastatic, dystrophic, and idiopathic; metastatic calcinosis is the most common type.
Vatsalya Kosuru   +6 more
openaire   +3 more sources

A Possible Link between Gastric Mucosal Atrophy and Gastric Cancer after Helicobacter pylori Eradication. [PDF]

open access: yesPLoS ONE, 2016
The effect of H. pylori eradication in gastric cancer prevention can be attributed to the improvement of atrophic gastritis, which is a known risk of gastric cancer. However, gastric cancer has also been diagnosed after long-term H.
Tomomitsu Tahara   +8 more
doaj   +1 more source

The Gastric Microbiome Communities and Endoscopic Mucosal Morphologies Associated with Premalignant Conditions

open access: yesMicroorganisms
The risk of Helicobacter pylori (H. pylori)-related gastric tumorigenesis is closely associated with the degree of chronic gastritis, although other gastric mucosa microbes may be relevant in this process.
Takuya Shijimaya   +9 more
doaj   +1 more source

Interaction of HS1BP3 with cortactin modulates TKS5 localisation, cell secretion and cancer malignancy

open access: yesMolecular Oncology, EarlyView.
Here, we demonstrate that HS1BP3 interacts with Cortactin through a proline‐rich region (PRR3.1) and show that this interaction, and HS1BP3 itself, promote cancer cell proliferation and invasion. Inhibition of this interaction leads to build‐up of TKS5 in multivesicular endosomes and altered secretion of CD63 and CD9, providing an explanation for the ...
Arja Arnesen Løchen   +9 more
wiley   +1 more source

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