Results 141 to 150 of about 40,581,100 (305)
2015/2016 UNMV General Science Assessment
, 2018 https://digitalrepository.unm.edu/provost_assessment/1048/thumbnail ...
UNMV General Science
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Molecular Oncology, EarlyView.The cell‐autonomous roles of interferon type 1 vary based on duration and intensity. While acute, robust signaling results in cancer cell cytotoxic effects, sustained, low‐level signaling is associated with tumor‐promoting effects. This review summarizes current research of IFN‐1 in cancer and within the clinical setting, with an emphasis on female ...
Ashlyn Conant +7 more
wiley +1 more source
XVIII Ibero-American Science and Technology Education Consortium General Assembly
, 2011 This eBook is a compendium of papers presented at the XVIII General Assembly of the Iberoamerican Science and Technology Education Consortium (ISTEC), held in Porto Alegre, Brazil, May 16-20, 2011.
Ibero-American Science and Technology Education Consortium
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Molecular Oncology, EarlyView.Tumour heterogeneity and clonal evolution of metastatic salivary gland cancer were evaluated in two patients with adenoid carcinoma and one patient with myoepithelial carcinoma. Radiology‐guided autopsy enabled multi‐region sampling (total samples n = 149), followed by whole‐genome sequencing and phylogenetic reconstruction (17 tumour samples, 4–7 per ...
Gerben Lassche +10 more
wiley +1 more source
2014/2015 UNMV General Science Assessment
, 2018 https://digitalrepository.unm.edu/provost_assessment/1020/thumbnail ...
UNMV General Science
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Molecular Oncology, EarlyView.This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi +6 more
wiley +1 more source
Molecular Oncology, EarlyView.Arginine methylation can be viewed as a persistence‐prone post‐translational modification regulated by a network of PRMTs. Competitive and compensatory interactions among PRMTs can redistribute methylation across substrate pools shaped by sequence, structural, spatial, and environmental layers, reinforcing RNA‐processing, chromatin, and signaling ...
So Hyun Kwon, Ji Min Lee
wiley +1 more source
Mechanisms and therapeutic opportunities of the ribotoxic stress response in cancer
Molecular Oncology, EarlyView.Cancer cells' high translational demand creates opportunities to therapeutically target ribosome function. Ribosome stalling and collisions activate ZAKα and the ribotoxic stress response (RSR), which can trigger rapid, p53‐independent apoptosis in cancer.
Anastassiya Kim +7 more
wiley +1 more source
, 2019 1 online resource (12 pages)Pursuant to Public act no. 18-125, section 5; "Monday, March 25, 2019.
Connecticut. General Assembly. General Law Committee. Student Data Privacy Task Force, author
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