Results 291 to 300 of about 17,207,173 (387)
INCOME‐GENERATING FUNCTIONS IN A LOW INCOME COUNTRY: COLOMBIA
Gary S. Fields, Tanja Schultz
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This article advocates integrating temporal dynamics into cancer research. Rather than relying on static snapshots, researchers should increasingly consider adopting dynamic methods—such as live imaging, temporal omics, and liquid biopsies—to track how tumors evolve over time.
Gautier Follain+3 more
wiley +1 more source
On the universal sequence generated by a class of unimodal functions
Li Wang, Nicholas D. Kazarinoff
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Interplay of integrins and selectins in metastasis
Here we review the role of integrins and their interplay with selectins in metastasis. The efficacy of integrin‐targeted therapies may be reduced in tumors where metastasis relies heavily on selectins. In certain tumors, integrins and selectins exhibit a synergistic interaction during intraperitoneal dissemination.
Diana Maltseva+2 more
wiley +1 more source
Pluripotent Stem Cells: Recent Advances and Emerging Trends. [PDF]
Wang AYL, Aviña AE, Liu YY, Kao HK.
europepmc +1 more source
On sieved orthogonal polynomials. IV. Generating functions
Mourad E. H. Ismail
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Inhibitor of DNA binding‐1 is a key regulator of cancer cell vasculogenic mimicry
Elevated expression of transcriptional regulator inhibitor of DNA binding 1 (ID1) promoted cancer cell‐mediated vasculogenic mimicry (VM) through regulation of pro‐angiogenic and pro‐cancerous genes (e.g. VE‐cadherin (CDH5), TIE2, MMP9, DKK1). Higher ID1 expression also increased metastases to the lung and the liver.
Emma J. Thompson+11 more
wiley +1 more source
From <i>Biomphalaria glabrata</i> to <i>Drosophila melanogaster</i> and <i>Anopheles gambiae</i>: the diversity and role of FREPs and Dscams in immune response. [PDF]
Li H+12 more
europepmc +1 more source
Path generating functions and continued fractions
I. P. Goulden, D. M. Jackson
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Alectinib resistance in ALK+ NSCLC depends on treatment sequence and EML4‐ALK variants. Variant 1 exhibited off‐target resistance after first‐line treatment, while variant 3 and later lines favored on‐target mutations. Early resistance involved off‐target alterations, like MET and NF2, while on‐target mutations emerged with prolonged therapy.
Jie Hu+11 more
wiley +1 more source