Results 201 to 210 of about 67,299 (230)
Some of the next articles are maybe not open access.
Related searches:
Related searches:
Complementation testing and genetic classification of Fanconi's anaemia
Lancet, The, 1994H Joenje, Fre Arwert, L P Ten Kate
exaly +3 more sources
Improved media for testing the mating reaction and genetic complementation of Ustilago hordei
Canadian Journal of Botany, 1992The sexual cycle of Ustilago hordei, which results in the formation of teliospores, requires growth on its barley host for completion. However, the early steps of mating, including conjugation and the formation of dikaryotic mycelium, can occur on artificial media.
Alfredo D. Martinez-Espinoza +3 more
openaire +1 more source
Mutation research, 1989
A collection of 81 his-3 mutants of Neurospora crassa was analyzed in assays for allelic complementation and specific revertibility. In these studies, the linearity of the complementation map of the his-3 cistron (Webber, 1965) was confirmed and mutants were classified as complementing with non-polarized or polarized complementation patterns, or non ...
L K, Overton +2 more
openaire +2 more sources
A collection of 81 his-3 mutants of Neurospora crassa was analyzed in assays for allelic complementation and specific revertibility. In these studies, the linearity of the complementation map of the his-3 cistron (Webber, 1965) was confirmed and mutants were classified as complementing with non-polarized or polarized complementation patterns, or non ...
L K, Overton +2 more
openaire +2 more sources
Birth Defects Research Part A: Clinical and Molecular Teratology, 2006
AbstractBACKGROUND: Nonsyndromic cleft lip (CL) with or without cleft palate (CLP) is a common human birth defect with complex genetic etiology. One of the unidentified genes maps to chromosome 17q21. A mouse strain, A/WySn, has CLP with complex genetic etiology that models the human defect, and 1 of its causative genes, clf1, maps to a region ...
Diana M, Juriloff +4 more
openaire +2 more sources
AbstractBACKGROUND: Nonsyndromic cleft lip (CL) with or without cleft palate (CLP) is a common human birth defect with complex genetic etiology. One of the unidentified genes maps to chromosome 17q21. A mouse strain, A/WySn, has CLP with complex genetic etiology that models the human defect, and 1 of its causative genes, clf1, maps to a region ...
Diana M, Juriloff +4 more
openaire +2 more sources
Vox Sanguinis, 1977
Abstract. Using the indirect immunofluorescence technique, P antigen is not detected on skin fibroblasts from Pk1, Pk2, or p donors. P antigen is reexpressed on polykaryon cells obtained by fusion of Pk ′ p fibroblasts. On the contrary, P antigen is not expressed on polykaryon cells from Pk ′ Pk or p ′ p fusion, All these data suggest that in Pk and p
M, Fellous +3 more
openaire +2 more sources
Abstract. Using the indirect immunofluorescence technique, P antigen is not detected on skin fibroblasts from Pk1, Pk2, or p donors. P antigen is reexpressed on polykaryon cells obtained by fusion of Pk ′ p fibroblasts. On the contrary, P antigen is not expressed on polykaryon cells from Pk ′ Pk or p ′ p fusion, All these data suggest that in Pk and p
M, Fellous +3 more
openaire +2 more sources
1986
Genetic polymorphisms of several components of human complement have been successively elucidated by some investigators1)~8) and now the existence of genetic polymorphism of the complement receptor as well as of the other components is being demonstrated.
K. Suzuki +5 more
openaire +1 more source
Genetic polymorphisms of several components of human complement have been successively elucidated by some investigators1)~8) and now the existence of genetic polymorphism of the complement receptor as well as of the other components is being demonstrated.
K. Suzuki +5 more
openaire +1 more source
Mutation research, 1989
Genetic fine structure analysis of the ad-3 and immediately adjacent genetic regions was made by means of complementation tests on all possible pairwise combinations of 50 X-ray-induced irreparable adenine-3 mutants (designated ad-3IR). All mutants were induced in either heterokaryon 11 or heterokaryon 12 of Neurospora crassa, 2-component heterokaryons
openaire +2 more sources
Genetic fine structure analysis of the ad-3 and immediately adjacent genetic regions was made by means of complementation tests on all possible pairwise combinations of 50 X-ray-induced irreparable adenine-3 mutants (designated ad-3IR). All mutants were induced in either heterokaryon 11 or heterokaryon 12 of Neurospora crassa, 2-component heterokaryons
openaire +2 more sources