Results 11 to 20 of about 50,967 (156)
Advances in Human Biology, 2021 Introduction: Hyperbilirubinemia is a common complication among neonates. The objective of this cross-sectional study was the prevalence of glucose 6-phosphate dehydrogenase (G6PD) deficiency among neonates with hyperbilirubinemia in the west of Iran ...
Fatemeh Eghbalian +3 more
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Glucose-6-phosphate Dehydrogenase Deficiency: A Review
International Journal of Medical Students, 2020 Deficiency of glucose-6-phosphate dehydrogenase enzyme is a common X-linked disorder that affects humans globally. It was first identified in the 1950s as a disorder that primarily affects the red blood cells causing a myriad of symptoms including acute
Nidhruv Ravikumar, Graeme Greenfield
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Acquired Glucose-6-Phosphate Dehydrogenase Deficiency
Journal of Clinical Medicine, 2022 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a hereditary condition caused by mutations on chromosome X and is transmitted by a sex-linked inheritance. However, impairment of G6PD activity may result from biochemical mechanisms that are able to inhibit the enzyme in specific clinical conditions in the absence of a structural gene-level defect.
Giovanni Mario Pes, Maria Pina Dore
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Kufa Journal for Nursing Sciences, 2022 Background: Glucose-6-phosphate dehydrogenase deficiency is a genetic disorder that affects red blood cells, which carry oxygen from the lungs to tissues throughout the body.
Sadiq Sabhan Mosa
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Glucose-6-phosphate dehydrogenase deficiency in Nigerian children. [PDF]
PLoS ONE, 2013 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common human enzymopathy and in Sub-Saharan Africa, is a significant cause of infection- and drug-induced hemolysis and neonatal jaundice.
Olatundun Williams +5 more
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Glucose-6-phosphate dehydrogenase (G6PD) Deficiency [PDF]
Iranian Journal of Public Health, 2008 Glucose-6-phosphate dehydrogenase (G6PD) Deficiency is the most prevalent enzymopathy in mankind. It has sex-linked inheritance. This enzyme exists in all cells. G6PD deficiency increases the sensitivity of red blood cells to oxidative damage.
DD Farhud , L Yazdanpanah
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Equine Glucose-6-phosphate Dehydrogenase Deficiency [PDF]
Veterinary Pathology, 1994 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a well-characterized X-linked inherited disorder in humans but has not been reported in horses. We describe a persistent hemolytic anemia and hyperbilirubinemia due to a severe G6PD deficiency in an American Saddlebred colt.
S L, Stockham, J W, Harvey, D A, Kinden
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Glucose-6-phosphate dehydrogenase deficiency [PDF]
Postgraduate Medical Journal, 1994 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most commonly known inherited disorder in man, and is estimated to affect 400 million people worldwide.' The highest prevalence rates (with gene frequencies in the range of 5-25% are found in tropical Africa, the Middle East, tropical and sub-tropical Asia, some parts of the Mediterranean, and ...
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The inorganic pyrophosphatase activity of erythrocytes in favism
The Turkish Journal of Pediatrics, 1961 Inorganic pyrophosphatase activity of erythrocytes was found to be decreased in subjects affected by the glucose-6-phosphate dehydrogenase deficiency. During the acute hemolytic episode of favism, pyrophosphatase activity shows a further critical fall.
Güngör Ernişli, Faolo Brunetti
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Geriatric Care, 2022 In glucose-6-phosphate-dehydrogenase deficiency (favism), exposure to oxidant agents can trigger hemolytic crises. The intravenous administration of very high doses of vitamin C was proposed as a treatment for severe coronavirus disease 2019 (COVID-19 ...
Filippo Luca Fimognari +6 more
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