Results 261 to 270 of about 105,092 (319)
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Studies of Respiration in H. pylori
2003In the procedures described here, respiratory metabolism is estimated by direct determination of oxygen uptake, measured as a reduction in the dissolved oxygen tension (DOT) of cell suspensions or extracts. Continuous monitoring of DOT is achieved using a Clark-type oxygen electrode linked to a chart recorder.
R J, Miles, H T, Chang
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Helicobacter pylori (H. pylori) Eradication
2017In 1998, Gasbarrini et al. reported that eradication therapy for Helicobacter pylori (H. pylori) increased platelet counts in most H. pylori-positive immune thrombocytopenia (ITP) patients. A large number of retrospective and prospective studies have since been performed in order to evaluate the efficacy of eradication therapy.
Toshiro Takafuta, Kingo Fujimura
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Therapy for H. pylori infection
Current Gastroenterology Reports, 1999The discovery that peptic ulcers can be caused by gastroduodenal infection with Helicobacter pylori and cured by eradication of this bacterial presence has made a profound impact on ulcer management. This article reviews the principles behind such treatment and the regimens in current use, including proton pump inhibitor triple therapies and ranitidine
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The Role of H. pylori Infection in Diabetes
Current Diabetes Reviews, 2005Helicobacter pylori [H. pylori], one of the most common chronic infections worldwide, is the main etiologic agent of gastritis, peptic ulcer and gastric cancer. Patients with diabetes mellitus are often affected by chronic infections. Many studies have evaluated the prevalence of H.
Ojetti, Veronica +5 more
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A Quintuple Therapy for H. pylori Eradication
The American Journal of Gastroenterology, 2007N ...
Zullo, A, Lorenzetti, R, Hassan, Cesare
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Basic bacteriology of H. pylori: H. pylori colonization factors
1994Helicobacter pylori is an unusual bacterial pathogen. This organism infects its host most probably in childhood and remains in most persons for life unless there has been specific H. pylori antimicrobial therapy or the stomach becomes an inhospitable environment due to such events as changes in the stomach due to long-term gastritis or surgery.
A. Lee, H. Mitchell
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1994
Helicobacter pylori-associated gastroduodenal inflammation is now considered to be the dominant factor in H. pylori-associated gastroduodenal ulcer formation. Healing the gastroduodenal mucosa through H. pylori eradication leads to a dramatic reduction in gastroduodenal ulcer relapse. Eradication of H.
G. N. J. Tytgat, L. A. Noach
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Helicobacter pylori-associated gastroduodenal inflammation is now considered to be the dominant factor in H. pylori-associated gastroduodenal ulcer formation. Healing the gastroduodenal mucosa through H. pylori eradication leads to a dramatic reduction in gastroduodenal ulcer relapse. Eradication of H.
G. N. J. Tytgat, L. A. Noach
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2003
Helicobacter pylori has been widely studied since its discovery in 1982 by Marshall and Warren (1), but many aspects of its structure, metabolism, and physiology, including its specific growth requirements, are still largely unknown, The organism is generally grown in complex media containing tissue extracts (e.g., of brain or heart) or proteolytic ...
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Helicobacter pylori has been widely studied since its discovery in 1982 by Marshall and Warren (1), but many aspects of its structure, metabolism, and physiology, including its specific growth requirements, are still largely unknown, The organism is generally grown in complex media containing tissue extracts (e.g., of brain or heart) or proteolytic ...
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1994
Helicobacter pylori therapy can either succeed (cure the infection) or fail. Failure is demonstrated by return of the original infecting organism soon after ending antimicrobial therapy (recrudescence of the infection). Cure of the infection does not guarantee that the individual will not be re-exposed to the bacterium and become reinfected with either
D. Y. Graham, R. M. Genta
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Helicobacter pylori therapy can either succeed (cure the infection) or fail. Failure is demonstrated by return of the original infecting organism soon after ending antimicrobial therapy (recrudescence of the infection). Cure of the infection does not guarantee that the individual will not be re-exposed to the bacterium and become reinfected with either
D. Y. Graham, R. M. Genta
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