Results 11 to 20 of about 90,454 (285)

Derivation of Escherichia coli O157:H7 from its O55:H7 precursor. [PDF]

open access: yesPLoS ONE, 2010
There are 29 E. coli genome sequences available, mostly related to studies of species diversity or mode of pathogenicity, including two genomes of the well-known O157:H7 clone. However, there have been no genome studies of closely related clones aimed at
Zhemin Zhou   +9 more
doaj   +5 more sources

Cefotaxime-, Ciprofloxacin-, and Extensively Drug-Resistant Escherichia coli O157:H7 and O55:H7 in Camel Meat

open access: yesFoods, 2023
The present study aimed to explore for the first time the occurrence and the antimicrobial resistance profiles of E. coli O157:H7 and O55:H7 isolates in camel meat in Egypt.
Khalid Ibrahim Sallam   +6 more
doaj   +4 more sources

Escherichia coli O157 : H7 colonization in cattle following systemic and mucosal immunization with purified H7 flagellin [PDF]

open access: yesInfection and Immunity, 2008
Escherichia coli O157:H7 is an important pathogen of humans. Cattle are most frequently identified as the primary source of infection, and therefore, reduction in E.
Smith, David George Emslie   +41 more
core   +4 more sources

Immunization with H7-HCP-tir-intimin significantly reduces colonization and shedding of Escherichia coli O157:H7 in goats. [PDF]

open access: yesPLoS ONE, 2014
Enterohemorrhagic Escherichia coli (EHEC) O157:H7 is the causative agent of hemorrhagic colitis and hemolytic uremic syndrome in humans. However, the bacterium can colonize the intestines of ruminants without causing clinical signs.
Xuehan Zhang   +3 more
doaj   +2 more sources

Inhibition of influenza H7 hemagglutinin-mediated entry.

open access: yesPLoS ONE, 2013
The recent outbreak of H7N9 influenza in China is of high concern to public health. H7 hemagglutinin (HA) plays a critical role in influenza entry and thus HA presents an attractive target for antivirals.
Michael Caffrey (151843)   +9 more
core   +5 more sources

Escherichia coli O157:H7

open access: yesThe Lancet, 1998
Escherichia coli O157 was first identified as a human pathogen in 1982. One of several Shiga toxin-producing serotypes known to cause human illness, the organism probably evolved through horizontal acquisition of genes for Shiga toxins and other virulence factors. E.
Mead, Paul S., Griffin, Patricia M.
openaire   +3 more sources

Analysis of the Role of Vaccinia Virus H7 in Virion Membrane Biogenesis with an H7-Deletion Mutant [PDF]

open access: yesJournal of Virology, 2013
ABSTRACT Essential vaccinia virus genes are often studied with conditional-lethal inducible mutants. Here, we constructed a deletion mutant lacking the essential H7R gene (the ΔH7 mutant) with an H7-expressing cell line. Compared to an inducible H7 mutant, the ΔH7 mutant showed a defect at an earlier step of virion membrane biogenesis, before
Xiangzhi, Meng   +4 more
openaire   +2 more sources

Genetically similar strains of Escherichia coli O157:H7 isolated from sheep, cattle and human patients [PDF]

open access: yes, 2012
Conclusions: The same pathogenic subtypes of VTEC O157:H7, including the highly virulent clade 8, appear to be present in both sheep and cattle in Sweden, suggesting strains can circulate freely between ruminant ...
Söderlund, Robert   +9 more
core   +1 more source

Generation of gene deletions and gene replacements in Escherichia coli O157:H7 using a temperature sensitive allelic exchange system [PDF]

open access: yes, 2006
In this work we describe protocols for the generation of gene deletions and gene replacements using a temperature sensitive plasmid in Escherichia coli O157:H7.
James R. Emmerson   +6 more
core   +1 more source

Transforming Growth Factor-β3 Regulates Adipocyte Number in Subcutaneous White Adipose Tissue

open access: yesCell Reports, 2018
Summary: White adipose tissue (WAT) mass is determined by adipocyte size and number. While adipocytes are continuously turned over, the mechanisms controlling fat cell number in WAT upon weight changes are unclear.
Paul Petrus   +19 more
doaj   +1 more source

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