Results 1 to 10 of about 389,551 (274)

Tissue damage in the heart after cardiac arrest induced by asphyxia and hemorrhage in newborn pigs [PDF]

open access: yesPediatric Research, 2019
Asphyxia of newborns is a severe and frequent challenge of the peri- and postnatal period.Forty-four neonatal piglets underwent asphyxia and hemorrhage (AH), followed by resuscitation with blood or crystalloid transfusion. In this study, 15 piglets (blood n = 9, NaCl n = 6, mean age 31 h) were randomly chosen.
Helmut Hummler   +10 more
openaire   +4 more sources

Enhanced Activity by NKCC1 and Slc26a6 Mediates Acidic pH and Cl− Movement after Cardioplegia-Induced Arrest of db/db Diabetic Heart

open access: yesMediators of Inflammation, 2019
Diabetic heart dysfunctions during cardiac surgeries have revealed several clinical problems associated with ion imbalance. However, the mechanism of ion imbalance mediated by cardioplegia and a diabetic heart is largely unclear. We hypothesized that ion
Minjeong Ji   +4 more
doaj   +2 more sources

Myocardial bridging presenting as myocardial ischaemia induced cardiac arrest: a case report

open access: yesBMC Cardiovascular Disorders, 2021
Background Myocardial bridging is a congenital anomaly defined as a segment of epicardial coronary arteries running through the myocardium. Various complications related to myocardial bridging have been reported, but at present, cardiac arrest has rarely
Young-Jae Ki
doaj   +2 more sources

Rat model of asphyxia-induced cardiac arrest and resuscitation

open access: yesFrontiers in Neuroscience, 2023
Neurologic injury after cardiopulmonary resuscitation is the main cause of the low survival rate and poor quality of life among patients who have experienced cardiac arrest.
Shuhang Yu   +4 more
doaj   +2 more sources

Phospholipid alterations in the brain and heart in a rat model of asphyxia-induced cardiac arrest and cardiopulmonary bypass resuscitation [PDF]

open access: yesMolecular and Cellular Biochemistry, 2015
Cardiac arrest (CA) induces whole-body ischemia, causing damage to multiple organs. Ischemic damage to the brain is mainly responsible for patient mortality. However, the molecular mechanism responsible for brain damage is not understood. Prior studies have provided evidence that degradation of membrane phospholipids plays key roles in ischemia ...
Joshua W Lampe   +4 more
openaire   +4 more sources

Glucose regulates the intrinsic inflammatory response of the heart to surgically induced hypothermic ischemic arrest and reperfusion

open access: yesPhysiological Genomics, 2017
We investigated the isolated working rat heart as a model to study early transcriptional remodeling induced in the setting of open heart surgery and stress hyperglycemia. Hearts of male Sprague Dawley rats were cold-arrested in Krebs-Henseleit buffer and subjected to 60 min normothermic reperfusion in the working mode with buffer supplemented with ...
Ahmed S. Bux   +4 more
openaire   +5 more sources

Main Complications of Mild Induced Hypothermia after Cardiac Arrest: A Review Article [PDF]

open access: yesJournal of Cardiovascular and Thoracic Research, 2014
The aim of the present study is to assess the complications of mild induced hypothermia (MIH) in patients with cardiac arrest. Presently, based on the guidelines of the American heart Association, MIH following successful cardiopulmonary resuscitation ...
Hassan Soleimanpour   +3 more
doaj   +2 more sources

Neonatal ketone body elevation regulates postnatal heart development by promoting cardiomyocyte mitochondrial maturation and metabolic reprogramming

open access: yesCell Discovery, 2022
Neonatal heart undergoes metabolic conversion and cell cycle arrest preparing for the increased workload during adulthood. Herein, we report that neonatal ketone body elevation is a critical regulatory factor for postnatal heart development.
Danyang Chong   +16 more
doaj   +2 more sources

Stroke Related Brain–Heart Crosstalk: Pathophysiology, Clinical Implications, and Underlying Mechanisms

open access: yesAdvanced Science
The emergence of acute ischemic stroke (AIS) induced cardiovascular dysfunctions as a bidirectional interaction has gained paramount importance in understanding the intricate relationship between the brain and heart.
Xuehui Fan   +8 more
doaj   +2 more sources

Catecholamine-Sensitive Right Ventricular Tachycardia in the Absence of Structural Heart Disease: A Mechanism of Exercise-Induced Cardiac Arrest

open access: yesCardiology, 1991
A case of exercise-induced cardiac arrest secondary to catecholamine-sensitive right ventricular tachycardia in the absence of apparent structural heart disease is presented. Amiodarone therapy prevented tachycardia induction, symptoms and clinical events despite a return to vigorous exercise.
Richard Taylor   +2 more
openaire   +4 more sources

Home - About - Disclaimer - Privacy