Results 211 to 220 of about 2,184,778 (369)
Cardiac endogenous transmitter system: molecular features, functions, and clinical implications. [PDF]
Xie D, Liang D, Zhou L, Liu Y, Chen YH.
europepmc +1 more source
From the nationwide multicenter consortium, 8,163 patients undergoing drug‐eluting stent implantation were classified according to the presence of the CYP2C19 loss‐of‐function (LoF) allele: rapid or normal metabolizers (RMs/NMs) vs. intermediate or poor metabolizers (IMs/PMs), and clinical risk was stratified using the CHADS‐P2A2RC and TRS 2°P scores ...
Hyun Woong Park +22 more
wiley +1 more source
B cells promote atrial fibrillation via autoantibodies. [PDF]
Yamazoe M +24 more
europepmc +1 more source
Major autonomic pathways to the atria and S-A and A-V nodes of the canine heart.
W. Geis, M. Kaye, W. C. Randall
semanticscholar +1 more source
Pregabalin and duloxetine are widely prescribed non‐opioid medications for chronic musculoskeletal pain. Pregabalin may increase the risk of heart failure, and duloxetine increases heart rate and blood pressure; however, little is known about their comparative cardiovascular safety.
Sachalee Campbell +11 more
wiley +1 more source
Automatic volumetric estimates of the left and right atrium using dynamic PET. [PDF]
Harms HJ +9 more
europepmc +1 more source
Carfilzomib is highly effective in the treatment of multiple myeloma, but it has been associated with cardiovascular adverse events that impact patient outcomes. Our prior global metabolomic analyses indicated an association between hydrophilic bile acids and carfilzomib‐cardiotoxicity risk, although a causal relationship remained to be determined ...
Samia Shabnaz +15 more
wiley +1 more source
FIBRILACIÓN AURICULAR EN EL PACIENTE HIPERTENSO. PAPEL DE LA AURÍCULA IZQUIERDA / Atrial fibrillation in hypertensive patients. Role of the left atrium [PDF]
Elibet Chávez González
doaj
Abstract Background Sporadic venous malformation (VM) is associated with the hyperactivating p.L914F mutation in TIE2, a receptor tyrosine kinase essential for vascular development. This mutation is not found in hereditary VM, suggesting incompatibility with life when expressed during early vascular development.
Lindsay J. Bischoff +6 more
wiley +1 more source

