Results 211 to 220 of about 89,054 (257)
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Helicobacter Pylori

Scandinavian Journal of Gastroenterology, 1991
Helicobacter pylori is a unique pathogen and the leading cause of chronic gastric inflammation. For many individuals the organism is of low virulence, causing only mild inflammation and generating few, if any, dyspeptic symptoms. For those with more severe inflammation, H. pylori infection may be causal in the generation of dyspeptic symptoms.
G N, Tytgat, L, Noach, E A, Rauws
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Helicobacter pylori

Der Gastroenterologe, 2009
Helicobacter pylori is associated with various gastroduodenal diseases such as peptic ulcer, functional dyspepsia, MALT lymphoma and distal gastric cancer. Diagnosis of H. pylori can be established by non-invasive ((13C)urea breath test, stool antigen test, serology) and invasive (histology, rapid urease test, culture) tests.
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Lipid A in Helicobacter pylori

Infection and Immunity, 1992
Free lipid A of Helicobacter pylori was characterized with regard to chemical composition, reactivity with anti-lipid A antibodies, and activity in a Limulus lysate assay. The predominant fatty acids of H. pylori lipid A were 3-OH-18:0, 18:0, 3-OH-16:0, 16:0, and 14:0.
I, Mattsby-Baltzer   +4 more
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Helicobacter Pylori

Current Treatment Options in Gastroenterology, 1999
All infected patients with a peptic ulcer should be treated for H. pylori. The role of treating H. pylori in patients with undiagnosed dyspepsia or non-ulcer dyspepsia, those taking nonsteroidal anti-inflammatory medications, or with a family history of gastric cancer remains controversial.
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Treatment of Helicobacter pylori

Best Practice & Research Clinical Gastroenterology, 2007
Triple therapy, consisting of two antibiotics, clarithomycin and amoxicillin or metronidazole in combination with a proton pump inhibitor (PPI) has become the first-line option for infection with Helicobacter pylori and has been recommended at several consensus conferences.
K, Wolle, P, Malfertheiner
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Helicobacter pylori and apoptosis

2002
Publisher Summary Apoptosis associated with Helicobacter pylori (H. pylori) may be studied by one of the two approaches: either by staining human and animal gastric tissue sections, or by coculturing H. pylori with gastric cells in vitro. The advantage of a coculture system is that the target cell and bacterium may be readily manipulated, thus ...
Emilia Mia, Sordillo, Steven F, Moss
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No Helicobacter pylori, no Helicobacter pylori‐associated peptic ulcer disease

Alimentary Pharmacology & Therapeutics, 1995
SUMMARYVirtually all duodenal ulcers (DUs) and the vast majority of gastric ulcers (GUS) are the consequence of Helicobacter pylori‐associated inflammation. In DUs, the inflammation is maximal in the antrum and is associated with gastric metaplasia in the bulb. Gastrin homeostasis is disturbed by H.
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Immunoproteome of Helicobacter pylori

2002
Publisher Summary This chapter describes how two-dimensional electrophoresis (2-DE) techniques can be combined with various immunoblotting procedures and how such data sets from infected patients can be evaluated in a semiquantitative manner as a basis for the development of sensitive and specific diagnostic tests.
Jungblut, Peter R., Bumann, Dirk
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Treatment of Helicobacter pylori

Current Opinion in Gastroenterology, 2011
The article will give an overview on reasons for treatment failure and tries to show new concepts for Helicobacter pylori treatment.Several new treatment options or modifications of already established regimens have been introduced to overcome treatment failure. Antibiotic resistance to H. pylori is the key factor for treatment failure.
Michael, Selgrad, Peter, Malfertheiner
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Helicobacter pylori infection

Journal of Antimicrobial Chemotherapy, 1993
The discovery of Helicobacter pylori is arguably the most significant advance made in gastroduodenal pathology this century. It is the most important cause of chronic gastritis, and almost certainly the major aetiological factor responsible for duodenal ulcer and probably for gastric ulcer as well.
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