Results 11 to 20 of about 83,777 (85)

Intrafamilial genotyping of Helicobacter pylori from faecal DNA [PDF]

open access: yes, 2011
Helicobacter pylori infection, often acquired in early childhood, is a global cause of undernutrition, gastritis, peptic ulcer disease and gastric carcinoma. This study tested the feasibility of using H.
Malcolm, C.   +22 more
core   +1 more source

Caveolin-1 protects B6129 mice against Helicobacter pylori gastritis. [PDF]

open access: yes, 2013
Caveolin-1 (Cav1) is a scaffold protein and pathogen receptor in the mucosa of the gastrointestinal tract. Chronic infection of gastric epithelial cells by Helicobacter pylori (H. pylori) is a major risk factor for human gastric cancer (GC) where Cav1 is
Ivana Hitkova (401838)   +50 more
core   +2 more sources

Recent acquisition of Helicobacter pylori by Baka Pygmies [PDF]

open access: yes, 2013
Both anatomically modern humans and the gastric pathogen Helicobacter pylori originated in Africa, and both species have been associated for at least 100,000 years. Seven geographically distinct H.
Linz, B.   +49 more
core   +2 more sources

Mixed infection with cagA positive and cagA negative strains of Helicobacter pylori lowers disease burden in The Gambia. [PDF]

open access: yes, 2011
BACKGROUND: The prevalence of Helicobacter pylori including strains with putatively virulent genotypes is high, whereas the H. pylori-associated disease burden is low, in Africa compared to developed countries.
Douglas E. Berg   +51 more
core   +1 more source

Prediction of extracellular proteases of the human pathogen Helicobacter pylori reveals proteolytic activity of the Hp1018/19 protein HtrA [PDF]

open access: yes, 2008
Exported proteases of Helicobacter pylori (H. pylori) are potentially involved in pathogen-associated disorders leading to gastric inflammation and neoplasia. By comprehensive sequence screening of the H. pylori proteome for predicted secreted proteases,
Löwer, Martin   +20 more
core   +1 more source

Suppression of Helicobacter pylori reduces gastrin releasing peptide stimulated gastrin release in duodenal ulcer patients. [PDF]

open access: yes, 1992
Helicobacter pylori increases gastrin release in duodenal ulcer patients. This may be through disruption or changes in the mucus layer affecting the access of luminal stimulants to gastrin releasing cells.
Ghosh, P   +6 more
core   +5 more sources

The sodium-dependent D-glucose transport protein of Helicobacter pylori [PDF]

open access: yes, 2008
Helicobacter pylori is a gram-negative pathogenic microaerophile with a particular tropism for the mucosal surface of the gastric epithelium. Despite its obligatory microaerophilic character, it can metabolize D-glucose and/or D-galactose in both ...
Kim E. Bettaney   +21 more
core   +1 more source

Epigenetic dynamics of Helicobacter pylori-induced COX-2 activation [PDF]

open access: yes, 2009
Background and Aims: Cyclooxigenase (COX)-2 is over expressed in gastrointestinal neoplasia and Helicobacter pylori (H. pylori) infection is casually linked to gastric cancer. We undertook this study to investigate the mechanism mediating H.
Peluso, Silvia
core   +1 more source

Ethnic and geographic differentiation of helicobacter pylori within Iran [PDF]

open access: yes, 2010
The bacterium Helicobacter pylori colonizes the human stomach, with individual infections persisting for decades. The spread of the bacterium has been shown to reflect both ancient and recent human migrations. We have sequenced housekeeping genes from H.
Sadegh Massarrat   +60 more
core   +2 more sources

Age of the association between Helicobacter pylori and man [PDF]

open access: yes, 2012
When modern humans left Africa ca. 60,000 years ago (60 kya), they were already infected with Helicobacter pylori, and these bacteria have subsequently diversified in parallel with their human hosts. But how long were humans infected by H.
Linz, B.   +62 more
core   +1 more source

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