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Hemifacial spasm

Current Treatment Options in Neurology, 2004
Hemifacial spasm (HFS) is a peripheral movement disorder caused by direct or indirect compression or distortion of the root exit zone of the seventh cranial nerve, which is most commonly compressed by an arterial loop, but also may be compressed by a tumor, cyst, or aneurysm.
Lawrence W. Kemp, Stephen G. Reich
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Hemifacial Spasm

Journal of Neurosurgery, 1978
A review of the literature as well as recent surgical observations indicate that hemifacial spasm may be caused by normal or pathological vascular structures that cross-compress the facial nerve. The critical area of compression is invariably found at the brain stem exit zone of the seventh cranial nerve.
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Hemifacial spasm

2011
Hemifacial spasm (HFS) is a peripherally induced movement disorder causing clonic or tonic contractions of the facial muscles. HFS is usually unilateral and sporadic. It may be primary (mainly attributed to vascular compressions of the seventh cranial nerve in the posterior fossa) or secondary to facial nerve or brainstem damage.
ABBRUZZESE, GIOVANNI   +2 more
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Hemifacial spasm

Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova, 2020
Hemifacial spasm (HFS), or facial hemispasm, is a paroxysmal hyperkinetic disorder involving muscles innervated by the facial nerve, mainly on the one hand. The development of HFS is based on neurovascular conflict. However, it is impossible to explain the clinical manifestations of HFS only by nerve compression.
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Familial hemifacial spasm

Movement Disorders, 1989
AbstractWe present a family in which hemifacial spasm involving in all cases the left side of the face occurred in five persons in three generations. Blink reflexes recorded in two cases demonstrated an unexpected R1 component on the affected side during stimulation of the contralateral side.
Jacek Bojakowski   +2 more
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Hemifacial spasm in sleep

Neurology, 1986
We conducted polygraphic studies during wakefulness and all-night sleep in 13 patients with cryptogenic and 3 with postparalytic hemifacial spasm. The movements decreased progressively with deepening sleep stages, reaching lowest values in REM sleep. The reduction was inversely related to the severity of movements during wakefulness.
Marco Zucconi   +5 more
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Hemifacial spasm in a child

Child's Nervous System, 1991
Hemifacial spasm in a 10-year-old child is herein reported. Computed tomography and angiography were normal. Magnetic resonance imaging showed an anomalous vascular structure, probably a redundant loop arising from AICA or PICA, which was identified at surgery. The spasm, presumably due to vascular compression at the root entry zone of the right facial
M. Grisoli   +4 more
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Familial Hemifacial Spasm

Archives of Ophthalmology, 1990
Three male patients in successive generations of the same family developed acquired left-sided hemifacial spasm. Magnetic resonance imaging of the proband demonstrated a pontine vertebral artery anomaly near the root of the affected facial nerve. This family and a similar family that was recently presented illustrate several interesting features: (1 ...
John A. McCrary   +4 more
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Atypical Hemifacial Spasm

Acta Neurochirurgica, 1998
Among 155 cases of hemifacial spasm (HFS), the authors found two cases of atypical HFS (1.3%) in which spasm started with the orbicularis oris and buccinator muscles, and gradually spread upward to involve the orbicularis occuli muscle, whereas the reverse process is usually seen in cases of typical HFS. The compression site in cases of atypical HFS is
T Miyamoto, Hiroshi Ryu, Seiji Yamamoto
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Latanoprost in hemifacial spasm

Prostaglandins & Other Lipid Mediators, 2002
An empirical observation that the prostaglandin derivate latanoprost quited facial spasms in patients with glaucoma prompted us to study latanoprost in non-glaucomatous patients with hemifacial spasms [HFS]. This explorative trial followed an open-label, prospective treatment design.
Luc Crevits   +3 more
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