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Familial Hemifacial Spasm

Archives of Ophthalmology, 1990
Three male patients in successive generations of the same family developed acquired left-sided hemifacial spasm. Magnetic resonance imaging of the proband demonstrated a pontine vertebral artery anomaly near the root of the affected facial nerve. This family and a similar family that was recently presented illustrate several interesting features: (1 ...
J B, Carter   +4 more
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Atypical Hemifacial Spasm

Acta Neurochirurgica, 1998
Among 155 cases of hemifacial spasm (HFS), the authors found two cases of atypical HFS (1.3%) in which spasm started with the orbicularis oris and buccinator muscles, and gradually spread upward to involve the orbicularis occuli muscle, whereas the reverse process is usually seen in cases of typical HFS. The compression site in cases of atypical HFS is
H, Ryu, S, Yamamoto, T, Miyamoto
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Familial hemifacial spasm

Movement Disorders, 1994
AbstractTwo brothers developed hemifacial spasm at 63 and 70 years of age. Spasms occurred on the left and right sides of the face, respectively. Computed tomography scan and magnetic resonance imaging failed to show any abnormality. In addition, a third sibling reported a history of a peripheral facial palsy, which remitted spontaneously without ...
F, Micheli   +4 more
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Latanoprost in hemifacial spasm

Prostaglandins & Other Lipid Mediators, 2002
An empirical observation that the prostaglandin derivate latanoprost quited facial spasms in patients with glaucoma prompted us to study latanoprost in non-glaucomatous patients with hemifacial spasms [HFS]. This explorative trial followed an open-label, prospective treatment design.
Luc, Crevits   +3 more
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Familial hemifacial spasm

Movement Disorders, 1989
AbstractWe present a family in which hemifacial spasm involving in all cases the left side of the face occurred in five persons in three generations. Blink reflexes recorded in two cases demonstrated an unexpected R1 component on the affected side during stimulation of the contralateral side.
A, Friedman, Z, Jamrozik, J, Bojakowski
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Baclofen in hemifacial spasm

International Journal of Neuroscience, 1987
Six patients with idiopathic hemifacial spasm refractory to the usually employed medications were successfully treated with baclofen. Adverse side effects were found in only 1 patient (memory loss) which was controlled with administration of piracetam. We suggest that there is an association between stress and certain cases of hemifacial spasm and that
Reuven Sandyk   +2 more
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Hemifacial Spasm in Infancy

Archives of Ophthalmology, 1990
Three infants presented with the onset of hemifacial spasm after birth and at the age of 10 months. One patient was found to have occlusion of the straight sinus and large collateral veins at the base of the brain, presumably due to venous sinus thrombosis, supporting the concept of vascular compression of the facial nerve at its exit from the brain ...
U, Flüeler   +5 more
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Infantile Hemifacial Spasm

Archives of Neurology, 1976
A 6-week-old infant had recurrent contractions of the facial musculature on the left side, which continued throughout early childhood. Surgical exploration at 5 1/2 years of age revealed a ganglioneuroma of the fourth ventricle. Hemifacial spasm (HFS) in infancy and childhood suggests the possibility of serious intracranial pathologic findings.
J W, Langston, B R, Tharp
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Psychogenic Hemifacial Spasm

The Journal of Neuropsychiatry and Clinical Neurosciences, 2001
Facial spasms that distort facial expression are typically due to facial dystonia, tics, and hemifacial spasm (HFS). Psychogenic facial spasms, however, have not been well characterized. The authors sought to 1) determine prevalence of psychogenic facial spasm in patients referred for evaluation of HFS and 2) draw attention to clinical characteristics ...
Tan E.-K., Jankovic J.
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Hemifacial Spasm: A Review

Surgical Neurology, 1991
Hemifacial spasm can be diagnosed by observation and clinical history. It is thought to arise primarily from compression of the facial nerve at the pons, usually by an adjacent artery. Although many approaches to treatment have been tried, the most effective is microvascular decompression of the facial nerve at the pons.
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