Results 241 to 250 of about 59,400 (291)
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Hemifacial spasm in sleep

Neurology, 1986
We conducted polygraphic studies during wakefulness and all-night sleep in 13 patients with cryptogenic and 3 with postparalytic hemifacial spasm. The movements decreased progressively with deepening sleep stages, reaching lowest values in REM sleep. The reduction was inversely related to the severity of movements during wakefulness.
Marco Zucconi   +5 more
openaire   +3 more sources

Hemifacial spasm in a child

Child's Nervous System, 1991
Hemifacial spasm in a 10-year-old child is herein reported. Computed tomography and angiography were normal. Magnetic resonance imaging showed an anomalous vascular structure, probably a redundant loop arising from AICA or PICA, which was identified at surgery. The spasm, presumably due to vascular compression at the root entry zone of the right facial
M. Grisoli   +4 more
openaire   +3 more sources

Familial Hemifacial Spasm

Archives of Ophthalmology, 1990
Three male patients in successive generations of the same family developed acquired left-sided hemifacial spasm. Magnetic resonance imaging of the proband demonstrated a pontine vertebral artery anomaly near the root of the affected facial nerve. This family and a similar family that was recently presented illustrate several interesting features: (1 ...
John A. McCrary   +4 more
openaire   +3 more sources

Atypical Hemifacial Spasm

Acta Neurochirurgica, 1998
Among 155 cases of hemifacial spasm (HFS), the authors found two cases of atypical HFS (1.3%) in which spasm started with the orbicularis oris and buccinator muscles, and gradually spread upward to involve the orbicularis occuli muscle, whereas the reverse process is usually seen in cases of typical HFS. The compression site in cases of atypical HFS is
T Miyamoto, Hiroshi Ryu, Seiji Yamamoto
openaire   +2 more sources

Latanoprost in hemifacial spasm

Prostaglandins & Other Lipid Mediators, 2002
An empirical observation that the prostaglandin derivate latanoprost quited facial spasms in patients with glaucoma prompted us to study latanoprost in non-glaucomatous patients with hemifacial spasms [HFS]. This explorative trial followed an open-label, prospective treatment design.
Luc Crevits   +3 more
openaire   +3 more sources

A correlative analysis between inflammatory cytokines and trigeminal neuralgia or hemifacial spasm

Neurological Research, 2019
Background: It is necessary to understand the mechanism of trigeminal neuralgia (TN) and hemifacial spasm (HFS) in order to seek for an effective noninvasive remedy.
Mingxing Liu   +4 more
semanticscholar   +1 more source

Familial hemifacial spasm

Movement Disorders, 1989
AbstractWe present a family in which hemifacial spasm involving in all cases the left side of the face occurred in five persons in three generations. Blink reflexes recorded in two cases demonstrated an unexpected R1 component on the affected side during stimulation of the contralateral side.
Jacek Bojakowski   +2 more
openaire   +3 more sources

Baclofen in hemifacial spasm

International Journal of Neuroscience, 1987
Six patients with idiopathic hemifacial spasm refractory to the usually employed medications were successfully treated with baclofen. Adverse side effects were found in only 1 patient (memory loss) which was controlled with administration of piracetam. We suggest that there is an association between stress and certain cases of hemifacial spasm and that
Reuven Sandyk   +2 more
openaire   +3 more sources

Familial hemifacial spasm

Movement Disorders, 1994
AbstractTwo brothers developed hemifacial spasm at 63 and 70 years of age. Spasms occurred on the left and right sides of the face, respectively. Computed tomography scan and magnetic resonance imaging failed to show any abnormality. In addition, a third sibling reported a history of a peripheral facial palsy, which remitted spontaneously without ...
Javier Adi   +4 more
openaire   +3 more sources

Hemifacial Spasm in Infancy

Archives of Ophthalmology, 1990
Three infants presented with the onset of hemifacial spasm after birth and at the age of 10 months. One patient was found to have occlusion of the straight sinus and large collateral veins at the base of the brain, presumably due to venous sinus thrombosis, supporting the concept of vascular compression of the facial nerve at its exit from the brain ...
E Brett   +5 more
openaire   +3 more sources

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