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Psychogenic Hemifacial Spasm

The Journal of Neuropsychiatry and Clinical Neurosciences, 2001
Facial spasms that distort facial expression are typically due to facial dystonia, tics, and hemifacial spasm (HFS). Psychogenic facial spasms, however, have not been well characterized. The authors sought to 1) determine prevalence of psychogenic facial spasm in patients referred for evaluation of HFS and 2) draw attention to clinical characteristics ...
Eng-King Tan, Joseph Jankovic
openaire   +3 more sources

Pathogenesis of Hemifacial Spasm [PDF]

open access: possible, 2020
Hemifacial spasm (HFS), a hyperactive motor dysfunction of the facial nerve, is believed to be caused by vascular compression at the root exit zone (REZ) of the facial nerve, which offered the basis for microvascular decompression (MVD) to become the standard treatment for HFS [1–5].
Jae Sung Park, Min Ho Lee
openaire   +1 more source

Baclofen in Hemifacial Spasm

European Neurology, 1984
A 58-year-old woman with long-standing hemifacial spasm experienced dramatic relief of symptomatology while receiving baclofen (Lioresal) in a daily dosage of 37.5 mg. Clinical response was noted within 48 h and she remained asymptomatic during 12 months of treatment. No toxic or side-effects were observed.
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Pathophysiology of hemifacial spasm

1994
Hemifacial spasm (HFS) is characterized by attacks of spasm on one side of the face that usually begin as small contractions around the eye and that over several years increase in intensity while progressing down the face as the intensity of the spasm increases [4, 5].
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Hemifacial Spasm: A Review

Surgical Neurology, 1991
Hemifacial spasm can be diagnosed by observation and clinical history. It is thought to arise primarily from compression of the facial nerve at the pons, usually by an adjacent artery. Although many approaches to treatment have been tried, the most effective is microvascular decompression of the facial nerve at the pons.
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Infantile Hemifacial Spasm

Archives of Neurology, 1976
A 6-week-old infant had recurrent contractions of the facial musculature on the left side, which continued throughout early childhood. Surgical exploration at 5 1/2 years of age revealed a ganglioneuroma of the fourth ventricle. Hemifacial spasm (HFS) in infancy and childhood suggests the possibility of serious intracranial pathologic findings.
Tharp Br, Langston Jw
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An investigation into quality of life improvement in patients undergoing microvascular decompression for hemifacial spasm.

Journal of Neurosurgery, 2018
OBJECTIVE Hemifacial spasm (HFS) is a movement disorder characterized by involuntary spasms of the facial muscles, and it can negatively impact quality of life (QOL).
J. Lawrence   +5 more
semanticscholar   +1 more source

Hemifacial spasm: case report

East African Medical Journal, 2006
A 53 year old lady with diabetes mellitus presented with right hemi-facial spasm (HFS). Brain MRI Scan revealed extensive pan-sinusitis and mild bilateral mastoiditis. She responded well to intravenous ceftriaxone therapy and the hemifacial spasm resolved.
J Matende, JO Jowi, MI Macharia
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Imaging of hemifacial spasm

Neurochirurgie, 2018
Almost all primary hemifacial spasms are associated with one or more neurovascular conflicts, most often at the root exit zone in the immediate vicinity of the brainstem. Imaging has first to exclude a secondary hemifacial spasm and secondly to search for and characterize the responsible neurovascular conflict(s).
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Pitfalls in the Diagnosis of Hemifacial Spasm

The Laryngoscope, 1997
AbstractHemifacial spasm (HFS), a condition characterized by involuntary unilateral facial spasms, is a disabling disorder resulting in functional compromise, patient frustration, cosmetic deformity, and social embarrassment. Compression of the seventh nerve at the root entry zone via vascular loop is presently the most widely accepted causative theory.
John J. Zappia   +3 more
openaire   +3 more sources

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