Results 81 to 90 of about 60,147 (311)

Secondary or Symptomatic Hemifacial Spasm caused by a Tumor of Cerebellopontine Angle Case Report and Review of the Literature

open access: yesIranian Journal of Neurosurgery, 2015
Background & Importance: Primary hemifacial spasm is usually caused by microvascular compression of the facial nerve at its root exit zone at brainstem without any space-occupying pathology.
Mohammad-Javad Mirzayan   +3 more
doaj  

Cluster headache associated with secondary unilateral blepharospasm: A case report and review of the literature

open access: yesJournal of Ophthalmic & Vision Research, 2017
Purpose: Cluster headache is one of the most serious types of headache that is accompanied by autonomic parasympathetic symptoms. Its association with hemifacial spasm in the same side had been rarely reported. The aim of this report is describing a case
Abbas Bagheri   +5 more
doaj   +1 more source

Flanker Task Performance in Isolated Dystonia (Blepharospasm): A Focus on Sequential Effects

open access: yesBrain Sciences, 2020
Isolated dystonia manifests with involuntary muscle hyperactivity, but the extent of cognitive impairment remains controversial. We examined the executive functions in blepharospasm while accounting for motor symptom-related distractions as a factor ...
Max Pekrul   +4 more
doaj   +1 more source

Vascular loop in the cerebellopontine angle causing pulsatile tinnitus and headache [PDF]

open access: yes, 2014
Tinnitus is a common disorder, it can be classified as pulsatile and non-pulsatile or objective and subjective. Pulsatile tinnitus is less common than non-pulsatile and can be due to vascular tumour such as glomus or vascular abnormality. We presented an
Asma, A.   +3 more
core  

Differentiating Non-Motor Symptoms in Parkinson's Disease from Controls and Hemifacial Spasm [PDF]

open access: yes, 2013
10.1371/journal.pone.0049596PLoS ...
A Schrag   +35 more
core   +2 more sources

Computer Vision Technologies in Movement Disorders: A Systematic Review

open access: yesMovement Disorders Clinical Practice, Volume 12, Issue 9, Page 1229-1243, September 2025.
Abstract Background Evaluation of movement disorders primarily relies on phenomenology. Despite refinements in diagnostic criteria, the accuracy remains suboptimal. Such a gap may be bridged by machine learning and video technology, which permit objective, quantitative, non‐invasive motor analysis.
Pasquale Maria Pecoraro   +4 more
wiley   +1 more source

Transposition of posterior inferior cerebellar artery could have caused ischemic complication in microvascular decompression for hemifacial spasm

open access: yesInterdisciplinary Neurosurgery, 2019
Ischemic complications are generally rare complications in microvascular decompression surgery for hemifacial spasm. We report a case of posterior inferior cerebellar artery (PICA) infarction post-microvascular decompression. A 68-year old woman with a 2-
Naohide Fujita   +5 more
doaj   +1 more source

Botulinum toxin type A efficient treatment in patients with hemifacial spasm, Guantanamo 2018-2019

open access: yesRevista Información Científica, 2020
Introduction: no research records were found addressing the results of the application of botulinum toxin type A in patients with hemifacial spasm in Guantanamo province.
Talía Marzo-Ramírez   +1 more
doaj  

Health-related quality of life outcomes from botulinum toxin treatment in hemifacial spasm

open access: yesTherapeutic Advances in Neurological Disorders, 2017
Background: Although botulinum neurotoxin (BoNT) injections may alleviate involuntary muscle contractions in hemifacial spasm substantially, it is less clear whether the motor effect would translate into improvements of health-related quality of life (HR-
Daniel Weiss   +10 more
doaj   +1 more source

Botulinum Neurotoxins: History, Mechanism, and Applications. A Narrative Review

open access: yesJournal of Neurochemistry, Volume 169, Issue 8, August 2025.
Botulinum neurotoxins (BoNTs), produced by Clostridium botulinum, bind irreversibly to presynaptic cholinergic nerve terminals and exploit a zinc‐dependent mechanism to cleave SNARE proteins, blocking acetylcholine release and causing flaccid paralysis.
Arik Monash   +3 more
wiley   +1 more source

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