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Hereditary non-spherocytic hemolytic disease
The American Journal of Medicine, 1960Abstract Twenty patients with hereditary non-sphero-cytic hemolytic disease, with varying rates of hemolysis, are described. This familial disorder is characterized by the presence of an intra-corpuscular defect of the erythrocytes without definitive morphologic abnormalities. It is important to differentiate this disease from hereditary spherocytosis
M E, CONRAD, W H, CROSBY, D L, HOWIE
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2021
Perinatal Hemolytic Disease main cause is Rh alloimmunization, which is caused by the exposure of Rh(D) negative pregnant women to Rh(D) positive red blood cells, leading to the development of antibodies that crosses the placenta and cause fetal red blood cells destruction.
Mário Dias Corrêa Júnior +2 more
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Perinatal Hemolytic Disease main cause is Rh alloimmunization, which is caused by the exposure of Rh(D) negative pregnant women to Rh(D) positive red blood cells, leading to the development of antibodies that crosses the placenta and cause fetal red blood cells destruction.
Mário Dias Corrêa Júnior +2 more
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Postgraduate Medicine, 1966
Acquired hemolytic disease (AHD) is an auto-immune disease characterized primarily by a shortened life-span and rapid regeneration of red cells. Idiopathic or secondary AHD is suspected any time a chronically anemic patient also has a chronically elevated reticulocyte count. It is diagnosed by demonstrating the immune mechanism by the antiglobulin test
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Acquired hemolytic disease (AHD) is an auto-immune disease characterized primarily by a shortened life-span and rapid regeneration of red cells. Idiopathic or secondary AHD is suspected any time a chronically anemic patient also has a chronically elevated reticulocyte count. It is diagnosed by demonstrating the immune mechanism by the antiglobulin test
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Haptoglobin and Hemolytic Disease
JAMA: The Journal of the American Medical Association, 1981To the Editor.— The article by Marchand et al (1980;243:1909) provides too much hope for a resurrection of haptoglobin determinations as a reliable indicator of hemolytic diseases. First, the introduction of any new or renewed diagnostic measurement should be based on a reliable diagnosis.
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Hemochromatosis and Hemolytic Disease
Archives of Internal Medicine, 1980Many hypotheses, most of them wrong, deal with the duodenum and iron absorption. At present, we accept that the balance of iron between the organism and its environment is controlled not by excretion of inadvertent excess but by the intestine's intelligent restriction of the amount it absorbs from dietary sources.
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Hemolytic Anemia in Wilson's Disease
New England Journal of Medicine, 1967JAUNDICE is a common presentation of Wilson's disease and may precede the neurologic features by many years.1 , 2 Infective hepatitis is often invoked as an explanation of the initial episode, and Wilson's disease is rarely considered at that time. Subsequently, neurologic symptoms may lead to a correct diagnosis when such features as jaundice and ...
N, McIntyre +4 more
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The Journal of Pediatrics, 1962
Summary The serologic diagnosis of ABO hemolytic disease with the two stage direct antiglobulin test with the use of fresh serum is presented. In 127 infants who were incompatible with the mother in the ABO blood group system, 9 were clinically affected infants.
A.C. Chan, F. Chung, H.G. Keitel
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Summary The serologic diagnosis of ABO hemolytic disease with the two stage direct antiglobulin test with the use of fresh serum is presented. In 127 infants who were incompatible with the mother in the ABO blood group system, 9 were clinically affected infants.
A.C. Chan, F. Chung, H.G. Keitel
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Archives of Pediatrics & Adolescent Medicine, 1983
In Reply .—Dr Ozsoylu raised an interesting point regarding our article. We believe, however, that the patient whom we described did not have hemolytic disease. He had not been jaundiced at birth, and no evidence of hemolysis had been noted during the three-year follow-up. Moreover, the raised reticulocyte count was noted only once, and we assumed that
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In Reply .—Dr Ozsoylu raised an interesting point regarding our article. We believe, however, that the patient whom we described did not have hemolytic disease. He had not been jaundiced at birth, and no evidence of hemolysis had been noted during the three-year follow-up. Moreover, the raised reticulocyte count was noted only once, and we assumed that
openaire +1 more source