Results 221 to 230 of about 694,423 (327)

Review Article: Liver Transplantation for Hepatocellular Carcinoma in the Era of Immune Checkpoint Inhibitors

open access: yesAlimentary Pharmacology &Therapeutics, Volume 62, Issue 6, Page 585-601, September 2025.
ICI is now the first‐line therapy for unresectable HCC and has revolutionised the management of advanced HCC and is now emerging as a modality for downstaging or bridging. The ideal timing of transplant after ICI should be at least 52 days. ABSTRACT Background Ablation, surgical resection and liver transplantation (LT) are curative therapies for ...
Anand V. Kulkarni   +2 more
wiley   +1 more source

Adult Left Liver Transplantation With Mesocaval Shunt With Porto‐Mesenteric Disconnection: Long‐Term Outcome

open access: yesClinical Transplantation, Volume 39, Issue 9, September 2025.
ABSTRACT Aiming to decrease portal venous pressure and to minimize the risk of small‐for‐size syndrome when using a partial liver graft for liver transplantation (LT), surgical techniques modulating venous portal inflow have been proposed. We report here our experience on the long‐term outcome after adult left split LT with mesocaval shunt (MCS) with ...
Jérôme Dumortier   +6 more
wiley   +1 more source

Rifaximin-α in Patients With Recurrent Episodes of Hepatic Encephalopathy Due to Cirrhosis Reduces Healthcare Utilization. [PDF]

open access: yesUnited European Gastroenterol J
van Doorn DJ   +10 more
europepmc   +1 more source

Predicting Clinical Outcomes of Cirrhosis Patients With Hepatic Encephalopathy From the Fecal Microbiome

open access: yesCellular and Molecular Gastroenterology and Hepatology, 2019
C. Sung   +13 more
semanticscholar   +1 more source

Mitochondrial Trifunctional Protein Deficiency due to HADHA Variants Masquerading as Charcot–Marie–Tooth Disease

open access: yesJournal of the Peripheral Nervous System, Volume 30, Issue 3, September 2025.
ABSTRACT Background and Aims Mitochondrial trifunctional protein deficiency (MTPD) is an inherited disorder of fatty acid β‐oxidation caused by mutations in HADHA or HADHB genes. It typically presents with cardiomyopathy or hepatic failure in early childhood; however, it may rarely present in adulthood with the neuromyopathic form.
Farkhanda Qaiser   +6 more
wiley   +1 more source

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