Results 111 to 120 of about 9,194,995 (243)
CEACAM1 participation in breast cancer progression
In invasive breast cancer (BC), CEACAM1 shifts from an apical to a uniform membranous/cytoplasmic pattern, or is lost, as tumors dedifferentiate, inversely tracking the Ki‐67 proliferative index. In MCF‐7 cells, only CEACAM1‐4L suppresses proliferation, repressing cell cycle and growth factor genes.
Mykola Lyndin +3 more
wiley +1 more source
Tumour heterogeneity and clonal evolution of metastatic salivary gland cancer were evaluated in two patients with adenoid carcinoma and one patient with myoepithelial carcinoma. Radiology‐guided autopsy enabled multi‐region sampling (total samples n = 149), followed by whole‐genome sequencing and phylogenetic reconstruction (17 tumour samples, 4–7 per ...
Gerben Lassche +10 more
wiley +1 more source
Modbury Hospital Annual Report 1997
Printed newsletter - the Modbury Hospital Annual report for ...
Modbury Hospital
core
Modbury Hospital planning photo 1
Black and white photo of 3 people looking at a file outside Modbury Hospital, possibly plans for future ...
Modbury Hospital
core +1 more source
'The Tracker' - Modbury Hospital News
A newsletter for Modbury Hospital that is of a different nature from the annual ...
Modbury Hospital
core +1 more source
Conferencias anatomoclínicas: caso no. veintinueve
Se hace una revisión de un caso clínico presentado en el Hospital San Juan de Dios ...
San Juan de Dios Hospital
doaj
This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi +6 more
wiley +1 more source
Black and white photo of the information desk for Modbury Hospital in the ...
Modbury Hospital
core +1 more source
Conferencias anatomoclínicas: caso no. veintitres
Se hace una revisión de un caso clínico presentado en el Hospital San Juan de Dios, Bogotá.
Hospital San Juan de Dios
doaj
We show that emergence of castration‐resistant prostate (CRPC) is associated with significant upregulation of cyclins that positively regulate cyclin‐dependent kinase 2 (CDK2) and concomitant downregulation of CDK4 cyclins. This renders CRPC cells dependent on the high activity of CDK2, and CDK2 inhibitors synergistically sensitize CRPC cells to both ...
Joyeeta Chatterjee +3 more
wiley +1 more source

