Results 81 to 90 of about 182,556 (397)

Late onset hydrocephalus in children with tuberculous meningitis

open access: yesJournal of Family Medicine and Primary Care, 2016
Hydrocephalus is a known complication of tuberculous meningitis (TBM). It is almost always present in patients who have had the disease for four to six weeks.
Disha Sharma, Ira Shah, Sharad Patel
doaj   +1 more source

A metabolite profile reveals the presence of neurodegenerative conditions according to severity of hydrocephalus [PDF]

open access: yes, 2018
Introduction: In obstructive congenital hydrocephalus, cerebrospinal fluid accumulation is associated with high intracranial pressure (ICP), ischemia/hypoxia, metabolic impairment, neuronal damage and astrocytic reaction.
Domínguez-Pinos, Dolores   +7 more
core  

Characterization of lower urinary tract symptoms in patients with idiopathic normal pressure hydrocephalus [PDF]

open access: yes, 2017
AIMS: The purpose of this study was to evaluate lower urinary tract symptoms (LUTS) in idiopathic normal pressure hydrocephalus (iNPH). METHODS: Patients with new-onset iNPH were prospectively evaluated for LUTS via detailed history and physical, and ...
Ahlberg   +25 more
core   +2 more sources

Communicating hydrocephalus due to cerebral venous sinus thrombosis treated with ventriculoperitoneal shunt

open access: yesAnnals of Indian Academy of Neurology, 2012
Cerebral venous sinus thrombosis (CVT) is a rare cerebrovascular disease with variable presentation. CVT rarely causes hydrocephalus. Communicating hydrocephalus due to CVT is extremely rare.
Rahul T Chakor   +3 more
doaj   +1 more source

Synthesis of realistic fetal MRI with conditional Generative Adversarial Networks [PDF]

open access: yesarXiv, 2022
Fetal brain magnetic resonance imaging serves as an emerging modality for prenatal counseling and diagnosis in disorders affecting the brain. Machine learning based segmentation plays an important role in the quantification of brain development. However, a limiting factor is the lack of sufficiently large, labeled training data.
arxiv  

A Mechanism for Ventricular Expansion in Communicating Hydrocephalus [PDF]

open access: yes, 2009
This report investigates a new possible molecular mechanism for the pathogenesis of hydrocephalus. New research by Dr. Miles Johnston has found that the injection of anti beta_1 integrin antibodies into the ventricle of rats causes a drop in parenchymal ...
Begg, R., Wilkie, K.
core  

GABAergic neurons regulate lateral ventricular development via transcription factor Pax5 [PDF]

open access: yes, 2013
Postmortem studies have revealed a downregulation of the transcription factor Pax5 in GABAergic neurons in bipolar disorder, a neurodevelopmental disorder, raising the question whether Pax5 in GABAergic neurons has a role in normal brain development.
Asada   +34 more
core   +2 more sources

THE HYDROCEPHALUS OF POLIOMYELITIS [PDF]

open access: yesArchives of Pediatrics & Adolescent Medicine, 1918
So prominent is this symptom of hydrocephalus in the pathologic and clinical picture of poliomyelitis, and so important is it from the viewpoint of treatment, that it seemed advisable to speak of it at length, and at the same time to show the value of Macewen's sign in its recognition. In order to adequately discuss the hydrocephalus of poliomyelitis,
openaire   +3 more sources

A Closer Look at the Global Management of Spina Bifida: The Implementation of Endoscopic Third Ventriculostomy in the Treatment of Spina Bifida-Related Hydrocephalus in Africa [PDF]

open access: yes, 2018
Spina bifida, specifically myelomeningocele, is a debilitating neural tube defect that affects patients and families throughout the world. Traditional management and treatment methods are described, followed by an explanation of why this is often ...
Burckart, Caryssa
core   +1 more source

Glycine decarboxylase deficiency causes neural tube defects and features of non-ketotic hyperglycinemia in mice. [PDF]

open access: yes, 2015
Glycine decarboxylase (GLDC) acts in the glycine cleavage system to decarboxylate glycine and transfer a one-carbon unit into folate one-carbon metabolism. GLDC mutations cause a rare recessive disease non-ketotic hyperglycinemia (NKH).
Brosnan, JT   +9 more
core   +1 more source

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