Results 151 to 160 of about 9,393 (183)
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Carbamazepine-induced hyperammonemia
American Journal of Health-System Pharmacy, 2009A case of carbamazepine-induced hyperammonemia is presented.A 26-year-old man with bipolar disorder, seizures, and mild mental retardation secondary to a traumatic brain injury began treatment with carbamazepine for aggression and seizure control. After three weeks of carbamazepine therapy, the patient arrived at the emergency department (ED) with ...
Erin N, Adams +2 more
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The hyperinsulinism/hyperammonemia syndrome
Reviews in Endocrine and Metabolic Disorders, 2010The hyperinsulinism/hyperammonemia (HI/HA) syndrome is the second most common form of congenital hyperinsulinism (HI). Children affected by this syndrome have both fasting and protein sensitive hypoglycemia combined with persistently elevated ammonia levels.
Andrew A, Palladino, Charles A, Stanley
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Hyperammonemia, Bane of the Brain
Clinical Pediatrics, 2004Ammonia, normally produced from catabolism of amino acids, is a deadly neurotoxin. As such, the concentration of free ammonia in the blood is very tightly regulated and is exceeded by two orders of magnitude by its physiologic derivative, urea. The normal capacity for urea production far exceeds the rate of free ammonia production by protein catabolism
Robert M, Cohn, Karl S, Roth
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Treatment of Congenital Hyperammonemias
Enzyme, 2017A rapid recognition of congenital hyperammonemia, a clear diagnostic workup and institution of a combined treatment without delay, by restriction of nitrogen supply, adequate caloric supply, substitution of missing metabolites, and use of alternate routes of nitrogen excretion will help to control hyperammonemic crises and improve the prognosis.
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Glutamate transporters in hyperammonemia
Neurochemistry International, 2002Evidence suggests that increases in brain ammonia due to congenital urea cycle disorders, Reye Syndrome or liver failure have deleterious effects on the glutamate neurotransmitter system. In particular, ammonia exposure of the brain in vivo or in vitro preparations leads to alterations of glutamate transport.
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Archives of Neurology, 1990
A 39-year-old man and his 42-year-old sister, both vegetarians, had episodic confusion for many years, but their mental function was normal between those episodes. They were recently diagnosed with hyperornithinemia, hyperammonemia, and homocitrullinuria syndrome.
M, Tuchman, D S, Knopman, V E, Shih
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A 39-year-old man and his 42-year-old sister, both vegetarians, had episodic confusion for many years, but their mental function was normal between those episodes. They were recently diagnosed with hyperornithinemia, hyperammonemia, and homocitrullinuria syndrome.
M, Tuchman, D S, Knopman, V E, Shih
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Electroencephalographic changes and hyperammonemia
Electroencephalography and Clinical Neurophysiology, 1958Resume Sept patients souffrants de differentes maladies et dont l'etat de conscience variait entre un etat de vigilance normale jusqu'au coma profond ont montre des ondes ⪡ triphasiques ⪢ dans leur trace EEG. Tous les 7 malades avaient une perturbation commune, c'est-a-dire une elevation de l'ammonium sanguin. L'auteur tire les conclusions suivantes:
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Electroencephalographic changes in hyperammonemia
Electroencephalography and Clinical Neurophysiology, 1956Abstract 1. 1. Correlation of the electroencephalogram and the blood ammonia levels which have been obtained serially in two patients has been observed and the literature pertinent to this has been reviewed. 2. 2. The “critical” level of blood ammonia that corresponds with the first significant change in the electroencephalogram is 200 to 400
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