Results 151 to 160 of about 9,482 (191)

Hyperammonemia in Alzheimer's disease

American Journal of Psychiatry, 1985
Postprandial blood ammonia levels were significantly higher in 22 patients with Alzheimer's disease than in 37 control subjects. In the Alzheimer group, fasting blood ammonia levels were significantly higher in patients whose EEGs showed triphasic waves than in patients without this change.
M, Fisman, B, Gordon, V, Feleki, E, Helmes, J, Appell, K, Rabheru   +5 more
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Postictal transient hyperammonemia

The American Journal of Emergency Medicine, 2008
We report cases of several patients who were sent to our hospital because of seizure attacks. Transient hyperammonemia was noted. Many biochemistry abnormalities, such as prolactin and creatine kinase, may be found after an episode of seizures, but hyperammonemia seems to not have been reported before.
Kuan-Ting, Liu, Chen-San, Su
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Hyperammonemia in the Neonate with Hypoxia

1982
Inherited defects in enzymes of the urea cycle and those in metabolism of several organic acids and some amino acids can cause neonatal hyperammonemia (Shih, 1978). Ballard et al. (1978) described five newborns with respiratory distress who developed hyperammonemia, but exhibited normal activities of urea cycle enzymes.
Y, Sakaguchi, K, Yuge, M, Yoshino, F, Yamashita, T, Hashimoto   +4 more
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Hyperammonemia in Multiple Myeloma

Acta Haematologica, 2009
Two patients with multiple myeloma who appeared to be producing ammonia are reported. Both patients showed hyperammonemia and amino acid disturbances, such as a low Fischer ratio. One patient had Bence Jones protein (λ) type myeloma and became comatose, but the hyperammonemia and disturbance of consciousness were improved by chemotherapy for the ...
H, Matsuzaki, M, Uchiba, K, Yoshimura, M, Yoshida, Y, Akahoshi, K, Okazaki, K, Takatsuki   +6 more
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Valproate‐induced hyperammonemia

Annals of Neurology, 1982
AbstractA patient with carbamyl phosphate synthetase deficiency had four episodes of hyperammonemia, up to 226 μM, associated with valproate (VPA) treatment. These were accompanied by vomiting, lethargy, and coma. A group of epileptic patients receiving VPA remained asymptomatic but had significantly higher mean plasma ammonium levels when compared to ...
M L, Batshaw, S W, Brusilow
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Hyperammonemia and Perinatal Asphyxia

Pediatrics, 1981
In a recent article Goldberg et al1 presented data on 12 asphyxiated newborn infants with high ammonia levels concomitant with abnormal liver function tests. They pointed out that the liver dysfunction, probably secondary to the ischemic-hypoxic injury, might have contributed to ammonia elevation by decreasing urea biosynthesis.
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Hyperammonemia in Marasmic Children

Journal of Tropical Pediatrics, 1994
The amino acids citrulline, ornithine and arginine, total serum proteins, serum enzymes glutamic oxalacetic and glutamic pyruvic transaminases, blood ammonia and urea were measured in 20 marasmic children with manifest psychomotor changes, before and after nutritional rehabilitation, as well as in 10 healthy age-matched children.
M R, Morsy, H, Madina, S A, Sharaf, A T, Soliman, M M, Elzalabany, M A, Ramadan   +5 more
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Carbamazepine-induced hyperammonemia

American Journal of Health-System Pharmacy, 2009
A case of carbamazepine-induced hyperammonemia is presented.A 26-year-old man with bipolar disorder, seizures, and mild mental retardation secondary to a traumatic brain injury began treatment with carbamazepine for aggression and seizure control. After three weeks of carbamazepine therapy, the patient arrived at the emergency department (ED) with ...
Erin N, Adams, Alla, Marks, Mitsi H, Lizer   +2 more
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The hyperinsulinism/hyperammonemia syndrome

Reviews in Endocrine and Metabolic Disorders, 2010
The hyperinsulinism/hyperammonemia (HI/HA) syndrome is the second most common form of congenital hyperinsulinism (HI). Children affected by this syndrome have both fasting and protein sensitive hypoglycemia combined with persistently elevated ammonia levels.
Andrew A, Palladino, Charles A, Stanley
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Glutamate transporters in hyperammonemia

Neurochemistry International, 2002
Evidence suggests that increases in brain ammonia due to congenital urea cycle disorders, Reye Syndrome or liver failure have deleterious effects on the glutamate neurotransmitter system. In particular, ammonia exposure of the brain in vivo or in vitro preparations leads to alterations of glutamate transport.
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