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Pediatrics, 1970
Dr. Thomas H. Milby, et al., of the California Department of Public Health1 described a significant (p. < .01) seasonal variation in the incidence of neonatal nonhemolytic unconjugated hyperbilirubinemia, observed in a hospital in a small, predominantly agricultural Californian community during a 4-year observation period (1963-1966).
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Dr. Thomas H. Milby, et al., of the California Department of Public Health1 described a significant (p. < .01) seasonal variation in the incidence of neonatal nonhemolytic unconjugated hyperbilirubinemia, observed in a hospital in a small, predominantly agricultural Californian community during a 4-year observation period (1963-1966).
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Hyperbilirubinemia in Preterm Neonates
Clinics in Perinatology, 2016Preterm neonates with increased bilirubin production loads are more likely to sustain adverse outcomes due to either neurotoxicity or overtreatment with phototherapy and/or exchange transfusion. Clinicians should rely on expert consensus opinions to guide timely and effective interventions until there is better evidence to refine bilirubin-induced ...
Vinod K. Bhutani +2 more
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Hyperbilirubinemia in preterm infants in Japan: New treatment criteria
Pediatrics International, 2018In 1992, Kobe University proposed treatment criteria for hyperbilirubinemia in newborns using total serum bilirubin and serum unbound bilirubin reference values.
I. Morioka
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Pediatrics, 1969
I read with interest the obervations of Dr. Milby and his co-workers (Pediatrics 43:601, 1969) regarding seasonal variations in neonatal hyperbilirubinemia. The differences between the two hospitals in the yearly fluctuation of the bilirubin levels is intriguing and raises a question about possible etiology which might bear consideration.
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I read with interest the obervations of Dr. Milby and his co-workers (Pediatrics 43:601, 1969) regarding seasonal variations in neonatal hyperbilirubinemia. The differences between the two hospitals in the yearly fluctuation of the bilirubin levels is intriguing and raises a question about possible etiology which might bear consideration.
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Conjugated Hyperbilirubinemia in Children
Pediatrics In Review, 2012A variety of anatomic, infectious, autoimmune, and metabolic diseases can lead to conjugated hyperbilirubinemia, both in the newborn period and later in childhood. The pediatric practitioner is most likely to encounter conjugated hyperbilirubinemia in the neonatal period.It is crucial to maintain a high degree of suspicion for cholestasis in the ...
Cara L. Mack, David Brumbaugh
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Coincidence of Schizophrenia and Hyperbilirubinemia
Pharmacopsychiatry, 1991In a retrospective study, the bilirubin plasma concentrations of every patient admitted to the Psychiatric Hospital were collected. Patients suffering from liver disease, substance abuse, overt hemolysis, or increased liver enzymes were excluded. Schizophrenics showed a significantly higher incidence of hyperbilirubinemia than patients suffering from ...
N. Müller, Ackenheil M, Schiller P
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Green light‐emitting diode phototherapy for neonatal hyperbilirubinemia: Randomized controlled trial
Pediatrics International, 2019The main photochemical pathway in phototherapy for neonatal hyperbilirubinemia is the production and elimination (in bile or urine) of cyclobilirubin, which is a structural photoisomer of bilirubin, and which is most efficiently produced by green light ...
Toru Kuboi +10 more
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Phototherapy and Hyperbilirubinemia of the Premature
Archives of Pediatrics & Adolescent Medicine, 1970Phototherapy to prevent hyperbilirubinemia in the premature infant was tested with 47 light-treated and 51 control infants with birth weights of 1,250 to 1,500 gm (2 lb 10 oz to 3 lb 4 oz). All infants had a negative Coombs' test. Treated infants were continuously nursed under a light chamber for 24 hours to 6 days of age. Bilirubin equaled or exceeded
Alberto Schwartz, Joan E. Hodgman
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Drug Metabolism And Disposition, 2018
Neonatal hyperbilirubinemia and the onset of bilirubin encephalopathy and kernicterus result in part from delayed expression of UDP-glucuronosyltransferase 1A1 (UGT1A1) and the ability to metabolize bilirubin. It is generally believed that acute neonatal
Shujuan Chen, R. Tukey
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Neonatal hyperbilirubinemia and the onset of bilirubin encephalopathy and kernicterus result in part from delayed expression of UDP-glucuronosyltransferase 1A1 (UGT1A1) and the ability to metabolize bilirubin. It is generally believed that acute neonatal
Shujuan Chen, R. Tukey
semanticscholar +1 more source