Results 171 to 180 of about 20,336 (222)

Elevated FGF23 Despite Biochemical Control Reveals Hidden Mineral Dysregulation in Chronic Hypoparathyroidism. [PDF]

J Endocr Soc
Malagrinò M, Piazza A, Bisceglia N, Capra S, Cantone C, Pagotto U, Zavatta G.   +6 more
europepmc   +1 more source

Anuria Following Solitary Left Renal Vein Ligation: An Old Surgery Myth Is Rigorously Challenged. [PDF]

Cureus
Onuigbo MA, Balonfentse O.
europepmc   +1 more source

Persistent hyperparathyroidism after kidney transplantation in children. [PDF]

Ren Fail
Park PG, Han A, Ahn YH, Min S, Ha J, Kang HG, Lee HK.   +6 more
europepmc   +1 more source

C-C motif receptor 2 is a core profibrotic factor in uremic cardiomyopathy.

Dis Model Mech
Bao JF, Guo BC, Mo JJ, Zhu H, Jia N, Hou F, Liu Y, Li A.   +7 more
europepmc   +1 more source

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Hyperphosphatemia in Lactic Acidosis

New England Journal of Medicine, 1977
The initial clue to the presence of lactic acidosis is usually a physical sign such as Kussmaul breathing, a sudden change in mental status or the complex of signs associated with shock.
L R, O'Connor, K L, Klein, J E, Bethune
exaly   +3 more sources

Hyperphosphatemia and Lactic Acidosis

New England Journal of Medicine, 1978
exaly   +3 more sources

Enema-induced hyperphosphatemia

The American Journal of Medicine, 1985
Severe hyperphosphatemia and hypocalcemia developed following the administration of a single hypertonic sodium phosphate enema in an adult with mild chronic renal insufficiency.
M, Biberstein, B A, Parker
openaire   +2 more sources

Management of hyperphosphatemia

Hemodialysis International, 2006
AbstractHyperphosphatemia is a well recognized risk factor for cardiovascular mortality in dialysis patients. Despite advanced technology and regular and efficient dialysis treatment the prevalence of hyperphosphatemia is still high. The goal of normalization of serum phosphorus (iP) levels can only be reached by optimization of dialysis prescription ...
openaire   +2 more sources

Hyperphosphatemia and Tumoral Calcinosis

Annals of Internal Medicine, 1968
Abstract Two teenage negro brothers with chronic hyperphosphatemia, one of whom exhibited heterotopic calcifications around large joints, underwent studies of phosphate metabolism.
J F, Wilber, E, Slatopolsky
openaire   +2 more sources

Hyperphosphatemia and phosphate binders

American Journal of Health-System Pharmacy, 2005
The pathophysiology of hyperphosphatemia associated with end-stage renal disease and treatment with phosphate binders are discussed.Phosphorus is an essential element necessary for the normal function of the human body, required for skeletal construction and synthesis of DNA, proteins, and adenosine triphosphate.
Jason J, Schucker, Kristina E, Ward
openaire   +2 more sources