Results 181 to 190 of about 20,336 (222)
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Emerging drugs for hyperphosphatemia
Expert Opinion on Emerging Drugs, 2007Cardiovascular mortality is the leading cause of death in the uremic patient. Hyperphosphatemia is considered an independent risk factor associated with cardiovascular morbidity and mortality in dialysis patients. As phosphate control is not efficient with diet or dialysis, phosphate binders are commonly prescribed in patients with chronic renal ...
BELLINGHIERI, Guido +2 more
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Hyperphosphatemia in Renal Failure
Blood Purification, 2005The recent recognition that hyperphosphatemia is a strong predictor of survival on dialysis has rekindled interest in the regulation and control of serum phosphate. In incipient renal failure hyperphosphatemia is prevented by increased fractional renal phosphate excretion mediated via an increase in parathyroid hormone and the novel phosphaturic ...
Eberhard, Ritz, Marie-Luise, Gross
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Hyperphosphatemia in multiple myeloma
Annals of Hematology, 1994We report three cases of IgG kappa multiple myeloma with pseudohyperphosphatemia. The patients' serum calcium levels were normal, and the hyperphosphatemia was not related to impaired renal function. No hypoparathyroidism was found, and no exogenous phosphate preparation had been given.
S, Oren +3 more
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Clinical nephrology, 1977
Serum phosphorus concentrations are maintained within narrow limits in humans. In the extracellular fluid most of the phosphorus is present in the inorganic form and at the level of the glomerulus greater than 90% of PO4 is ultrafilterable. The kidney plays a key role in PO4 homeostasis. Micropuncture experiments have demonstrated that 60 to 70% of the
E, Slatopolsky +4 more
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Serum phosphorus concentrations are maintained within narrow limits in humans. In the extracellular fluid most of the phosphorus is present in the inorganic form and at the level of the glomerulus greater than 90% of PO4 is ultrafilterable. The kidney plays a key role in PO4 homeostasis. Micropuncture experiments have demonstrated that 60 to 70% of the
E, Slatopolsky +4 more
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Tumoral calcinosis with hyperphosphatemia
The Indian Journal of Pediatrics, 2005Tumoral calcinosis is a rare disorder of mineral metabolism among adolescents and young adults characterized by deposition of calcific masses around large joints. It is less commonly reported in pediatric population and commonly mistaken for bone tumors.
S, Mahadevan +2 more
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Pediatrics, 1963
Connelly et al., in their recent article on neonatal hyperphosphatemia (Pediatrics, 30:425, 1962), came to the conclusion that "an effect of the hormone was evident on day three, the clearance of the treated infants being significantly greater than that of the controls. . .
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Connelly et al., in their recent article on neonatal hyperphosphatemia (Pediatrics, 30:425, 1962), came to the conclusion that "an effect of the hormone was evident on day three, the clearance of the treated infants being significantly greater than that of the controls. . .
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Assessment of Hyperphosphatemia and Hypophosphatemia
Clinics in Laboratory Medicine, 1993Methodologic aspects including causes of factitious hyperphosphatemia and hypophosphatemia are summarized. The differential diagnosis of hyperphosphatemia is reviewed under its three broad causes: decreased glomerular filtration rate, increased exogenous or endogenous phosphate load, and increased renal tubular phosphate reabsorption.
E, Bourke, N, Yanagawa
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Hyperphosphatemia in a Burn Patient
Journal of Burn Care & Rehabilitation, 2001Hypophosphatemia has been observed in severely burned patients and has been associated with increased mortality. Hyperphosphatemia has rarely been described in this population. We present a burn patient with hyperphosphatemia, hypercalciuria, and suppressed parathyroid hormone level 5 months after the initial burn.
V D, Bachelder +2 more
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Hyperphosphatemia and Hypocalcemia in Lemurs
Journal of the American Veterinary Medical Association, 1978SUMMARY A progressive nutritional disease characterized by hyperphosphatemia, hypocalcemia, soft tissue mineralization, impaired locomotion, increased alkaline phosphatase activity, and poor skeletal mineralization developed in a small group of captive lemurs (Lemur catta and L variegatus).
F N, Tomson, R R, Lotshaw
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