Results 121 to 130 of about 1,745 (164)

Hypokalemic periodic paralysis

The Journal of Emergency Medicine, 1986
Generalized motor weakness is a common complaint in the emergency department and has a wide differential diagnosis that includes both organic and infectious etiologies. We report the case of a previously healthy young man with symptoms of muscular paralysis and associated hypokalemia.
L, Cannon, J, Bradford, J, Jones
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Hypokalemic periodic paralysis

Pediatric Emergency Care, 1988
We report two cases of hypokalemic periodic paralysis in children presenting to the emergency department with diffuse weakness and no antecedent history of neurologic disease. The literature is briefly reviewed. Any child with acute weakness and normal mental status should undergo serum potassium determination to allow prompt diagnosis and therapy.
T, Schiller, P S, Auerbach
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Hypokalemic Periodic Paralysis with Arrhythmia

New England Journal of Medicine, 1972
IN 1963, Klein and his co-workers1 described a variant of periodic paralysis in which intermittent attacks of muscular paralysis occurred in patients who also had cardiac arrhythmia.
L P, Levitt, L I, Rose, D M, Dawson
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Hypokalemic Periodic Paralysis or Hypokalemic Muscle Weakness?

Archives of Neurology, 1981
To the Editor— Bennet and Forman, in their recent report of a case of chronic toluene exposure that had caused tubular dysfunction, loss of potassium, and muscle weakness (Archives1980;37:673), used the term "hypokalemic periodic paralysis" to describe the disease.
O J, Buruma, J J, Schipperheyn
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Hypokalemic periodic paralysis exacerbated by acetazolamide

Neurology, 1981
Although acetazolamide usually prevents paralytic attacks in hypokalemic periodic paralysis, not all patients benefit from this treatment. We studied a father and two sons in whom attack frequency and severity increased on acetazolamide. Administration of triamterene virtually abolished attacks in three separate single-blind trials totaling more than ...
C F, Torres, R C, Griggs, R T, Moxley, A N, Bender   +3 more
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Hypokalemic Periodic Paralysis

Archives of Neurology, 1979
To the Editor.— Hypokalemic periodic paralysis has been treated in many ways, such as by the use of potassium supplements and spironolactone to increase the patient's level of potassium 1 and most recently by the use of acetazolamide. 2 This last method is thought not to alter potassium metabolism 3 but rather to work through the creation of metabolic
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Recognizing thyrotoxic hypokalemic periodic paralysis

JAAPA, 2018
ABSTRACT Thyrotoxic hypokalemic periodic paralysis (THPP) is a rare but potentially serious complication of thyrotoxicosis. The resulting muscle weakness is profound, associated with more severe hypokalemia, yet reversible. However, clinicians must be cautious because patients can develop life-threatening hyperkalemia during treatment ...
Kamini, Patel, Jonathan V, McCoy, Patrick M, Davis   +2 more
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Multiple Sclerosis and Hypokalemic Periodic Paralysis

Archives of Neurology, 1984
To the Editor. —In a recent issue of theArchives, Toglia et al 1 referred to an instance of the association of multiple sclerosis (MS) and hypokalemic paralysis in the same person, which they believed to be the first reported. A similar case history has, in fact, already been recorded from this department.
J, Braham, M, Sadeh
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The Effect of Acidosis in Hypokalemic Periodic Paralysis

Archives of Neurology, 1976
Metabolic acidosis was produced in two patients with hypokalemic periodic paralysis by the administration of ammonium chloride over a period of three days. The challenging test of glucose and insulin produced a substantially smaller reduction of both serum potassium concentration and muscle strength than when the patients were tested in normal acid ...
M A, Jarrell, M, Greer, T H, Maren
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HYPOKALEMIC PERIODIC PARALYSIS OF THYROTOXIC ORIGIN

Australian and New Zealand Journal of Medicine, 1987
Abstract A 50‐year‐old Vietnamese man suffered recurrent episodes of hypokalemic periodic paralysis during treatment for thyrotoxicosis. Suspected precipitants of the paralysis were oral prednisolone, strenuous exertion and poor compliance with medications. Propranolol prevented the periodic paralysis. Control of the thyrotoxicosis was complicated by a
J G, McHutchison, R A, Melick, J D, Wark
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