Results 61 to 70 of about 490,574 (151)

Hypokalemic Periodic Paralysis

open access: yesJournal of Islamic International Medical College, 2016
Hypokalemic Periodic Paralysis is a group of rare inherited disorders that can cause temporary and often recurrent episodes of acute flaccid paralysis. Several conditions e.g.
Abidullah Khan , Muhammad Farooq
doaj  

Periodic paralysis: clinical evaluation in 20 patients

open access: yesArquivos de Neuro-Psiquiatria, 1994
Twenty patients with periodic paralysis were evaluated and the aspects studied included epidemiological data, clinical manifestations, ancillary tests, treatment and evolution.
Célia Harumi Tengan   +2 more
doaj   +1 more source

Unmasking Idiopathic Inflammatory Myopathy: A Case of Proximal Weakness in a Young Male With Co‐Occurring Vitamin D Deficiency

open access: yesClinical Case Reports, Volume 13, Issue 11, November 2025.
ABSTRACT Idiopathic inflammatory myopathy (IIM) encompasses rare autoimmune disorders causing muscle inflammation and weakness, with subtypes including dermatomyositis, necrotizing myopathy, antisynthetase syndrome, and inclusion body myositis. This case report details a 25‐year‐old South Asian male presenting with a 60‐day history of progressive ...
Ibrahim Khalil   +3 more
wiley   +1 more source

Hypokalemic Periodic Paralysis as the First Manifestation of Thyrotropin-Secreting Pituitary Adenoma

open access: yesCase Reports in Endocrinology, 2019
Thyrotoxic periodic paralysis is an unusual neurological manifestation of thyrotoxicosis, and even rarer when it occurs in thyrotropin-secreting pituitary adenoma, only 6 cases having been previously reported. We describe a case of pituitary microadenoma
Chatchon Kaewkrasaesin   +4 more
doaj   +1 more source

Thyrotoxic Periodic Paralysis with Hypokalemia in an Adult Male from Nepal: A Case Report

open access: yesJournal of Nepal Medical Association, 2019
Thyrotoxic periodic paralysis is rare complication of hyperthyroidism characterized by the sudden onset of hypokalemia and muscle paralysis. It is typically present in young Asian males.
Sabina Khadka   +4 more
doaj   +1 more source

Sjögren's Syndrome With Distal Renal Tubular Acidosis and Hypokalemic Myopathy in Pregnancy: A Rare Case

open access: yesClinical Case Reports, Volume 13, Issue 11, November 2025.
Case timeline. ABSTRACT Sjogren's syndrome may rarely present with distal renal tubular acidosis (dRTA), causing severe hypokalemia and myopathy. A 27‐year‐old primigravida at 15 weeks gestation presented with progressive proximal weakness and myalgia.
Nabiha Khan   +5 more
wiley   +1 more source

Hypokalemic periodic paralysis: an unusual presentation of primary hyperparathyroidism

open access: yes, 2018
Primary hyperparathyroidism is an endocrine condition characterized by hyper secretion of parathyroid hormone (PTH). It has a wide varied clinical presentation from mild nonspecific symptoms to classical disease.
Rakesh Chandru K., Zahir Hussain S.
core   +1 more source

Hypokalemic Periodic Paralysis Due To Distal Renal Tubular Acidosis

open access: yes, 2015
Hypokalemic periodic paralysis (HPP) is a disorder that characterized by attacks of skeletal muscle paralysis depending on the changes in serum potassium levels, and can occur due to primary and secondary causes.
GÜLLÜ, MEHMET NEZİR   +5 more
core   +2 more sources

Hypokalemic Periodic Paralysis in a 10-year-old Child: Case Study

open access: yesВопросы современной педиатрии
Background. Hypokalemic periodic paralysis is a rare form of paralysis in children resulting from genetically determined electrolyte disorders. Its diagnosis is complicated due to transient nonspecific symptoms. Case description. The disease onset was at
Artem A. Ivanov   +3 more
doaj   +1 more source

Voltage sensor charge loss accounts for most cases of hypokalemic periodic paralysis

open access: yes
Background: Several missense mutations of CACNA1S and SCN4A genes occur in hypokalemic periodic paralysis. These mutations affect arginine residues in the S4 voltage sensors of the channel. Approximately 20% of cases remain genetically undefined. Methods:
Schorge S   +10 more
core   +5 more sources

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