Results 251 to 260 of about 30,396,780 (392)
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian +10 more
wiley +1 more source
Multi-lifecycle Assessment of Close-loop Recyclable Wind Turbine Blades. [PDF]
Pender K, Romoli F, Fuller J.
europepmc +1 more source
A comprehensive genomic and proteomic analysis of cervical cancer revealed STK11 and STX3 as a potential biomarkers of chemoradiation resistance. Our study demonstrated EGFR as a therapeutic target, paving the way for precision strategies to overcome treatment failure and the DNA repair pathway as a critical mechanism of resistance.
Janani Sambath +13 more
wiley +1 more source
Corrigendum to 'Psychosocial Impact of Dental Aesthetics Among Aboriginal and Torres Strait Islander peoples: Mixed-Methods Study Using Psychosocial Impact of Dental Aesthetics Questionnaire': [International Dental Journal Volume 76, Issue 1, February 2026, 109339]. [PDF]
Nath S +7 more
europepmc +1 more source
YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy +9 more
wiley +1 more source
Foam-Based Wearable Devices Embedded with Shear-Thickening Fluids for Biomedical Protective Applications. [PDF]
Oyetunji O, Zabihollah A.
europepmc +1 more source
The impact of supply chain integration on performance: A contingency and configuration approach
Barbara B. Flynn +2 more
semanticscholar +1 more source
Emerging role of ARHGAP29 in melanoma cell phenotype switching
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster +3 more
wiley +1 more source

