Results 221 to 230 of about 125,957 (306)
Translating cardiovascular ion channel and Ca2+ signalling mechanisms into therapeutic insights
Abstract figure legend This white paper integrates mechanistic discoveries across ion channel biology, Ca2+ signalling and multiscale cardiovascular physiology to highlight new opportunities for accelerating research and guiding next‐generation therapies. Printed with permission from ®Anita Impagliazzo Medical Illustration. [Correction added on 2 March
Silvia Marchianò +18 more
wiley +1 more source
Right posterior communicating artery aneurysm with multiple arterial stenoses and moyamoya disease: A case report. [PDF]
Liu J, Shi Z, Liu H, Wen G.
europepmc +1 more source
ABSTRACT Purpose Blood T1 is a key parameter for hemodynamic quantification in both non‐contrast‐ and contrast‐enhanced imaging. Individual vessel T1 has been measured using a modified Look–Locker scheme with multi‐shot EPI or FLASH in high spatial resolution, requiring ∼1 min.
Zechen Xu, Feng Xu, Qin Qin, Dan Zhu
wiley +1 more source
Combined IVIg and 500 mg/day IVMP achieve superior efficacy over monotherapies in anti‐LGI1 encephalitis, with optimal safety. Higher‐dose IVMP (1000 mg) accelerates response but increases adverse events, particularly in diabetic patients. Cognitive deficits persist post‐treatment, necessitating long‐term management.
Yao Tan +6 more
wiley +1 more source
Cerebral in situ ischemic postconditioning: From bench to bedside and beyond. [PDF]
Jiang X, Zhao W, Xu Y, Meng F, Wei M.
europepmc +1 more source
ABSTRACT Ravulizumab, a second‐generation complement component 5 inhibitor (C5i) derived from eculizumab, with improved pharmacokinetics, is the current standard‐of‐care treatment for patients with paroxysmal nocturnal hemoglobinuria (PNH), where available.
Alexander Röth +8 more
wiley +1 more source
Postoperative symptomatic vasospasm in pituitary surgery: case series and systematic review - a matter of blood? [PDF]
Sannwald LW +9 more
europepmc +1 more source
Objective Immunothrombosis contributes to ischemic stroke pathophysiology through neutrophil extracellular trap (NET) formation, which promotes thrombus stabilization and microvascular dysfunction. DNase1 is the principal endonuclease responsible for NET degradation.
B. Díaz‐Benito +10 more
wiley +1 more source

