Results 131 to 140 of about 785,029 (297)
Thromboembolic Complications Following So-called "Good Risk" Cases of Myocardial Infarction [PDF]
Adolfo Francisco, Irving S. Wright
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Mitochondrial transplantation induces macrophage polarization toward an anti‐inflammatory M2 phenotype, enhances their reparative capacities, and facilitates mitochondrial transfer to cardiomyocytes, collectively promoting tissue repair and functional recovery post‐myocardial infarction.
Yuning Zhang +10 more
wiley +1 more source
Effects of Papaverine upon Ectopic Ventricular Tachycardia Produced by Myocardial Infarction [PDF]
A. Sidney Harris +3 more
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PPP1R3B induces anti‐inflammatory M2 macrophage polarization and maintains energy supply in plaques. Its absence accelerates plaque progression. PPP1R3B regulates M2 macrophage polarization and energy metabolism via phosphorylated STAT3 (p‐STAT3), which plays a dual role by activating anti‐inflammatory transcriptional programs through the PPAR‐γ/PGC‐1α/
Lin Shen +12 more
wiley +1 more source
FMO2 Promotes Angiogenesis via Regulation of N‐Acetylornithine
This study identifies flavin‐containing monooxygenase 2 (FMO2) as a novel proangiogenic regulator in endothelial cells. Targeted FMO2 ablation impairs vessel sprouting, whereas its compensation potently enhances angiogenesis. Metabolomics and single‐cell sequencing reveal that FMO2 drives vascular growth via the N‐acetylornithine/ATF3/NOTCH1 axis ...
Jingyi Wang +15 more
wiley +1 more source
SEPTAL INFARCTION WITH RECOVERY OF THE ELECTROCARDIOGRAM [PDF]
D. G. Abrahams
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