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Infectious Arthritis in the Elderly [PDF]
, 2011 Infections have long been known to be leading causes of morbidity and mortality in the elderly population. Immunosenescence of both the innate and adaptive immune systems contributes largely to this and we have examined the studies which show changes in toll-like receptors (TLRs), cytokines, dendritic cells, antibody response, and T-cells.
Nicole Melendez, Luis R. Espinoza
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Infectious agents in reactive arthritis
Current Opinion in Rheumatology, 1993It is now clear that the deposition and persistence of bacterial antigens in the joint are significant features of reactive arthritis. It is possible that in some instances this represents persistence of live bacteria, and several studies point to the potential value of antimicrobial therapy.
Andrew Keat, Rod Hughes
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Treatment of Infectious Arthritis and Osteomyelitis
Veterinary Clinics of North America: Food Animal Practice, 1991Infectious arthritis and osteomyelitis are commonly encountered diseases of the skeletal system that can result in premature loss of an affected animal from the herd. Selection of the best possible treatment approach for an individual animal requires a solid understanding of the pathophysiologic processes involved in the infection of bone or joint ...
Ava M. Trent, Donald Plumb
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Surgical and conservative treatment of infectious arthritis in cattle.
Journal of the American Veterinary Medical Association, 1974During a 5-year period, conservative vs surgical treatment was studied in 81 cattle afflicted with infectious arthritis. Arthritis of the pedal joint and infectious polyarthritis were not included in the study. Surgical treatment included opening of the
F. Verschooten +5 more
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Best Practice & Research Clinical Rheumatology, 2006
In contrast to the outstanding achievements made in therapy for autoimmune arthritides, not least rheumatoid arthritis, the pace of progress in therapy for infectious arthritis remains slow. This has primarily to do with the complex task of, on the one hand, killing the invading microorganisms and, on the other, to down-regulate the exaggerated immune ...
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In contrast to the outstanding achievements made in therapy for autoimmune arthritides, not least rheumatoid arthritis, the pace of progress in therapy for infectious arthritis remains slow. This has primarily to do with the complex task of, on the one hand, killing the invading microorganisms and, on the other, to down-regulate the exaggerated immune ...
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Acute Infectious Arthritis [PDF]
, 1988 Acute infectious arthritis is a medical emergency. Delay in recognition and initiation of therapy can increase morbidity and permanent sequelae, particularly in infants, the elderly, and patients with major underlying diseases (7, 19). In some cases acute infectious arthritis may be associated with mortality.
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Radiology, 1925
The patient is an American woman, aged 60. Her mother died at the age of 76, of la grippe; her father, of pulmonary tuberculosis, at 51; brother, at 84, following operation, the nature of which is not known, and sister, at 22, of pulmonary tuberculosis. The patient was married at the age of 21; has one son, aged 35, in good health; no miscarriages. Her
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The patient is an American woman, aged 60. Her mother died at the age of 76, of la grippe; her father, of pulmonary tuberculosis, at 51; brother, at 84, following operation, the nature of which is not known, and sister, at 22, of pulmonary tuberculosis. The patient was married at the age of 21; has one son, aged 35, in good health; no miscarriages. Her
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Infectious agents in arthritis and autoimmunity
Modern Rheumatology, 2003Abstract The distinctions between infection, chronic arthritis, and autoimmune diseases have steadily blurred over the past decades. The proposed pathomechanisms underlying these interesting associations include putative pathways from infection to innate and adaptive immunity, molecular mimicry, and certain microbial and host factors.
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Infectious Etiology of Rheumatoid Arthritis
Rheumatic Disease Clinics of North America, 1991The infectious etiology of rheumatoid arthritis has been a long-standing hypothesis and in recent years is being examined with greater sophistication and scientific rigor. Synovitis may result indirectly from infection by the deposition of circulating immune complexes, by molecular mimicry, by in situ antigen deposition, or by arthritogenic toxins.
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