Results 251 to 260 of about 693,842 (409)

Intermittent ozone inhalation during house dust mite-induced sensitization primes for adverse asthma phenotype

open access: yesRedox Biology
The ability of air pollution to induce acute exacerbation of asthma is well documented. However, the ability of ozone (O3), the most reactive gaseous component of air pollution, to function as a modulator during sensitization is not well established ...
Salik Hussain   +9 more
doaj  

Validation of an AI-Powered Smart Dry Powder Inhaler (RS01X) for Asthma and COPD in a Clinical Setting. [PDF]

open access: yesInt J Chron Obstruct Pulmon Dis
Chamaon D   +3 more
europepmc   +1 more source

THE USE OF CO2 INHALATION AS A TEST OF CIRCULATION TIME 1 [PDF]

open access: bronze, 1939
Richard Gubner   +2 more
openalex   +1 more source

Biomimetic Microstructured Scaffold with Release of Re‐Modified Teriparatide for Osteoporotic Tendon‐to‐Bone Regeneration via Balancing Bone Homeostasis

open access: yesAdvanced Science, EarlyView.
This study develops RPTH by re‐modifying teriparatide and designs a biomimetic microstructured scaffold (BMRP) loaded with RPTH to address osteoporotic tendon‐to‐bone interface repair. The BMRP restores tissue architecture by balancing bone homeostasis and remodeling the microstructure, achieving functional recovery in an osteoporotic rotator cuff tear
Chengzhong Xu   +11 more
wiley   +1 more source

THE INHALATION OF HEXOESTROL [PDF]

open access: yesBritish Journal of Pharmacology and Chemotherapy, 1947
G. L. Foss, J. H. Gaddum
openaire   +3 more sources

Standardization of bronchial inhalation challenge procedures.

open access: yesJournal of Allergy and Clinical Immunology, 1975
H. Chai   +8 more
semanticscholar   +1 more source

Identification of CD38high Monocyte as a Candidate Diagnostic Biomarker and Therapeutic Target for Sepsis

open access: yesAdvanced Science, EarlyView.
This study uncovers that CD38high monocytes discriminate sepsis and sterile inflammation and are associated with 28‐day mortality in bacterial sepsis. Targeting CD38 therapy reduces inflammatory response in monocytes and in sepsis mice model. Mechanistically, CD38‐HIF‐1α/glycolysis/MGO loop exacerbates sepsis‐induced immune dysregulation.
Ning Hua   +19 more
wiley   +1 more source

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