Results 121 to 130 of about 7,374,554 (273)
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian +10 more
wiley +1 more source
Knowledge, barriers, attitude, perception, and ability of public and private institution graduating pharmacy students toward deprescribing and polypharmacy: a cross-sectional survey study in Northwest Ethiopia. [PDF]
Alemu MA +9 more
europepmc +1 more source
Gateways for Institutional-Grade Commerce and Interoperability of Digital Assets [PDF]
Rafael Belchior +3 more
openalex +1 more source
YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy +9 more
wiley +1 more source
Determinants of Educational Institution Enrolment Among Orphaned and Vulnerable Children in Namibia: A Multi-Year Analysis 2018-2024. [PDF]
Dzinamarira T +8 more
europepmc +1 more source
Student Aid and Institutional Autonomy: Congressional Decisions
Harry J. Hogan
openalex +2 more sources
Emerging role of ARHGAP29 in melanoma cell phenotype switching
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster +3 more
wiley +1 more source

