Results 241 to 250 of about 5,881,292 (359)
Catholic Institutions and Justice Obligations [PDF]
Flanigan, Rosemary, LaCroix, W. L.
core +1 more source
EMT‐associated bias in the Parsortix® system observed with pancreatic cancer cell lines
The Parsortix® system was tested for CTC enrichment using pancreatic cancer cell lines with different EMT phenotypes. Spike‐in experiments showed lower recovery of mesenchymal‐like cells. This was confirmed with an EMT‐inducible breast cancer cell line.
Nele Vandenbussche+8 more
wiley +1 more source
Creating codes of ethics for public health professionals and institutions. [PDF]
Thomas JC+7 more
europepmc +1 more source
Analysis of treatment‐naïve high‐grade serous ovarian carcinoma (HGSOC) and control tissues for ERVs, LINE‐1 (L1), inflammation, and immune checkpoints identified five clusters with diverse patient recurrence‐free survivals. An inflammation score was calculated and correlated with retroelement expression, where one novel cluster (Triple‐I) with high ...
Laura Glossner+6 more
wiley +1 more source
Building Public Health Quantitative Methods Capacity and Networks in sub-Saharan Africa: An Evaluation of a Faculty Training Program. [PDF]
Ntshebe O+7 more
europepmc +1 more source
Opportunities for Research in Botany Offered by American Institutions
Peter H. Raven
openalex +2 more sources
A DIA‐MS‐based proteomics analysis of serum samples from GB patients and healthy controls showed that high levels of IL1R2 and low levels of CRTAC1 and HRG in serum are associated with poor survival outcomes for GB patients. These circulating proteins could serve as biomarkers for the prediction of outcome in patients with GB.
Anne Clavreul+11 more
wiley +1 more source
Utilisation of routine health information system and associated factors among health workers in public health institutions of Gofa zone, South Ethiopia regional state:a mixed-methods study. [PDF]
Doka BK, Worku AG, Negeri KG, Kassa DH.
europepmc +1 more source
Roman Historical Sources and Institutions
Charles Upson Clark, Henry A. Sanders
openalex +2 more sources
Cotargeting EGFR and STAT3 with Erlotinib and TTI‐101 impairs both 2D and 3D growth of ETV1‐overexpressing prostate cancer cells by disrupting a self‐sustaining ETV1–EGFR positive feedback loop that promotes EGFR and STAT3 expression and phosphorylation (activation).
Elsa Gomes Paiva+5 more
wiley +1 more source