Results 71 to 80 of about 851,846 (204)

Conducted Interference, Challenges and Interference Cases

open access: yesIEEE Electromagnetic Compatibility Magazine, 2015
Conducted interference has become increasingly problematic in the past few years, especially within the 2-150 kHz band. The high penetration of non-linear loads, combined with distributed generation, will influence the voltage profile, i.e. the power quality. New technologies will introduce new types of interference.
openaire   +2 more sources

Redox regulation meets metabolism: targeting PRDX2 to prevent hepatocellular carcinoma

open access: yesMolecular Oncology, EarlyView.
PRDX2 acts as a central redox hub linking metabolic dysfunction‐associated steatohepatitis (MASH) to hepatocellular carcinoma (HCC). In normal hepatocytes, PRDX2 maintains redox balance and metabolic homeostasis under oxidative stress. In contrast, during malignant transformation, PRDX2 promotes oncogenic signaling, stemness, and tumor initiation ...
Naroa Goikoetxea‐Usandizaga   +2 more
wiley   +1 more source

Fonetične in ortografske interference pri govorjeni romunščini v Španiji

open access: yesVestnik za Tuje Jezike, 2012
Članek Fonetične in ortografske interference pri govorjeni romunščini v Španiji prikaže z dvojne (fonetične in ortografske) perspektive nekaj pojavov, ki so nastali pri romunsko španskih stikih.
Ioana Jieanu, Eugenia Baciu
doaj   +1 more source

RNA Interference [PDF]

open access: yesBioMed Research International, 2006
Sioud, Mouldy, Haoudi, Abdelali
openaire   +2 more sources

Tumour–host interactions in Drosophila: mechanisms in the tumour micro‐ and macroenvironment

open access: yesMolecular Oncology, EarlyView.
This review examines how tumour–host crosstalk takes place at multiple levels of biological organisation, from local cell competition and immune crosstalk to organism‐wide metabolic and physiological collapse. Here, we integrate findings from Drosophila melanogaster studies that reveal conserved mechanisms through which tumours hijack host systems to ...
José Teles‐Reis, Tor Erik Rusten
wiley   +1 more source

Cotargeting TREM2 and IL2 pathways triggers multipronged anticancer immunity

open access: yesMolecular Oncology, EarlyView.
Von Locquenghien et al. report that MiTE‐144, a triggering receptor expressed on myeloid cells 2 (TREM2) blocking antibody fused to interleukin‐2 (IL2) variant with tumour microenvironment restricted activation, demonstrates superior anticancer efficiency in a preclinical setting.
Isaure Vanmeerbeek   +2 more
wiley   +1 more source

Türkiye’s dangerous game in Cyprus: The 2020 presidential elections and the Turkish interference in North Cyprus

open access: yesJournal of Modern Science
Objectives While Türkiye’s attempts to interfere in the internal affairs of Turkish Cypriots are not a new phenomenon, the scale of the current one should cause concern to the international community. As is the apparent hardening of Türkiye’s negotiating
Przemysław Osiewicz, Wiesław Lizak
doaj   +1 more source

De quelques interférences caractéristiques de l’apprentissage de l’amazighe à l’école marocaine

open access: yesMultilinguales, 2015
In the context of the didactics of languages, our article aims at analyzing some interference errors committed by arabophone learners in the process of learning the Amazigh language in the Moroccan school.
Mohammed Nabih, Mounir Bourray
doaj   +1 more source

RIPK4 function interferes with melanoma cell adhesion and metastasis

open access: yesMolecular Oncology, EarlyView.
RIPK4 promotes melanoma growth and spread. RIPK4 levels increase as skin lesions progress to melanoma. CRISPR/Cas9‐mediated deletion of RIPK4 causes melanoma cells to form less compact spheroids, reduces their migratory and invasive abilities and limits tumour growth and dissemination in mouse models.
Norbert Wronski   +9 more
wiley   +1 more source

Engineered extracellular vesicles enriched with the miR‐214/199a cluster enhance the efficacy of chemotherapy in ovarian cancer

open access: yesMolecular Oncology, EarlyView.
Loss of the miR‐214/199a cluster is associated with recurrence in ovarian cancer. Engineered small extracellular vesicles (m214‐sEVs) elevate miR‐214‐3p/miR‐199a‐5p in tumor cells, suppress β‐catenin, TLR4, and YKT6 signaling, reprogram tumor‐derived sEV cargo, reduce chemoresistance and migration, and enhance carboplatin efficacy and survival in ...
Weida Wang   +12 more
wiley   +1 more source

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